Literature DB >> 7573230

Shear stress may stimulate release and action of nitric oxide in the human fetal-placental vasculature.

K M Wieczorek1, A S Brewer, L Myatt.   

Abstract

OBJECTIVE: Our purpose was to determine the effect of perfusate flow or viscosity on release and action of nitric oxide in the human fetal-placental vasculature in vitro. STUDY
DESIGN: Cotyledons from normal term placentas were perfused with Hanks' buffered saline solution gassed with 95% oxygen and 5% carbon dioxide, pH 7.4, at 37 degrees C with a maternal flow rate of 10 ml/min. Fetal flow rate was varied from 1 to 10 ml/min at 10-minute intervals (n = 6), and perfusion pressure was recorded. Viscosity was varied by perfusion of both circulations with Hanks' balanced salt solution containing 0, 2.5, 5.0, 7.5, or 10% dextran, the concentration being changed every 20 minutes and pressure recorded at fetal flow rates of 4 (n = 4) or 1 ml/min (n = 7). All experiments were performed in the presence or absence of the nitric oxide synthase inhibitor N-nitro-L-arginine (10(-3) mol/L) in the same placentas. Data were analyzed by analysis of variance with repeated measures.
RESULTS: Increasing fetal flow rate with or without N-nitro-L-arginine resulted in a significant increase in perfusion pressure (p = 0.0011). Addition of N-nitro-L-arginine gave an overall significant increase in perfusion pressure (p = 0.0048). At a fetal flow rate of 4 ml/min increasing dextran concentration with or without N-nitro-L-arginine gave a significant increase in perfusion pressure (p = 0.0011), but the increase in perfusion pressure in the presence of N-nitro-L-arginine did not reach significance (p = 0.06). At 1 ml/min increasing dextran concentration resulted in a significant increase in fetal perfusion pressure (p = 0.001), but no significant effect of N-nitro-L-arginine was observed.
CONCLUSION: Altering shear stress by increasing fetal flow rate or viscosity of the medium may increase synthesis and release of nitric oxide, which attenuates increases in perfusion pressure.

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Year:  1995        PMID: 7573230     DOI: 10.1016/0002-9378(95)90327-5

Source DB:  PubMed          Journal:  Am J Obstet Gynecol        ISSN: 0002-9378            Impact factor:   8.661


  7 in total

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Authors:  B J Krause; M A Hanson; P Casanello
Journal:  Placenta       Date:  2011-07-27       Impact factor: 3.481

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Review 4.  Physiological mechanisms of vascular response induced by shear stress and effect of exercise in systemic and placental circulation.

Authors:  Iván Rodríguez; Marcelo González
Journal:  Front Pharmacol       Date:  2014-09-16       Impact factor: 5.810

5.  Emerging concepts of shear stress in placental development and function.

Authors:  L C Morley; D J Beech; J J Walker; N A B Simpson
Journal:  Mol Hum Reprod       Date:  2019-06-06       Impact factor: 4.025

6.  Nitric oxide synthase activity in human trophoblast, term placenta and pregnant myometrium.

Authors:  J Al-Hijji; Ellika Andolf; Ricardo Laurini; Satish Batra
Journal:  Reprod Biol Endocrinol       Date:  2003-06-28       Impact factor: 5.211

7.  Piezo1 channels are mechanosensors in human fetoplacental endothelial cells.

Authors:  L C Morley; J Shi; H J Gaunt; A J Hyman; P J Webster; C Williams; K Forbes; J J Walker; N A B Simpson; D J Beech
Journal:  Mol Hum Reprod       Date:  2018-10-01       Impact factor: 4.025

  7 in total

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