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133 in total

1. Adenovirus-mediated expression of tissue plasminogen activator does not alter endothelial cell proliferation and invasion.

Authors: D K Zablocki; J J Rade; B R Alevriadou
Journal: In Vitro Cell Dev Biol Anim Date: 2000 Nov-Dec Impact factor: 2.416

Review 2. Cholesterol lowering and coronary artery disease: mechanisms of risk reduction.

Authors: R A Archbold; A D Timmis
Journal: Heart Date: 1998-12 Impact factor: 5.994

3. Macrophage metalloproteinases degrade high-density-lipoprotein-associated apolipoprotein A-I at both the N- and C-termini.

Authors: Ivano Eberini; Laura Calabresi; Robin Wait; Gabriella Tedeschi; Angela Pirillo; Lina Puglisi; Cesare R Sirtori; Elisabetta Gianazza
Journal: Biochem J Date: 2002-03-15 Impact factor: 3.857

Review 4. Molecular and cellular mechanisms of myocardial remodeling.

Authors: Melanie Maytin; Wilson S Colucci
Journal: J Nucl Cardiol Date: 2002 May-Jun Impact factor: 5.952

5. Molecular cloning and characterization of a matrix metalloproteinase, from Caenorhabditis elegans: employed to identify homologous protein from Angiostrongylus cantonensis.

Authors: Rui Sun; Zheng-yu Li; Han-jiang He; Jie Wei; Juan Wang; Qi-xian Zhang; Jia Zhao; Xi-mei Zhan; Zhong-dao Wu
Journal: Parasitol Res Date: 2011-12-14 Impact factor: 2.289

Matrix metalloproteinases and cardiovascular disease.

1. Adenovirus-mediated expression of tissue plasminogen activator does not alter endothelial cell proliferation and invasion.

Review 2. Cholesterol lowering and coronary artery disease: mechanisms of risk reduction.

3. Macrophage metalloproteinases degrade high-density-lipoprotein-associated apolipoprotein A-I at both the N- and C-termini.

Review 4. Molecular and cellular mechanisms of myocardial remodeling.

5. Molecular cloning and characterization of a matrix metalloproteinase, from Caenorhabditis elegans: employed to identify homologous protein from Angiostrongylus cantonensis.

Review 6. Clinical implications of matrix metalloproteinases.

Review 7. Coronary arterial remodeling: from bench to bedside.

8. Chronic hyperhomocysteinemia causes vascular remodelling by instigating vein phenotype in artery.

9. QM/MM Studies of the Matrix Metalloproteinase 2 (MMP2) Inhibition Mechanism of (S)-SB-3CT and its Oxirane Analogue.

10. Plasmin inhibition increases MMP-9 activity and decreases vein wall stiffness during venous thrombosis resolution.