Literature DB >> 7536416

Apoptosis and functional Fas antigen in rheumatoid arthritis synoviocytes.

T Nakajima1, H Aono, T Hasunuma, K Yamamoto, T Shirai, K Hirohata, K Nishioka.   

Abstract

OBJECTIVE: To determine whether apoptosis occurs in rheumatoid arthritis (RA) synoviocytes, and if this phenomenon is dependent on the Fas/Apo-1 pathway.
METHODS: Apoptotic change in vivo was examined in RA synovial cells by several standard methods. The ability of cells to undergo Fas-induced apoptosis was determined in vitro.
RESULTS: Typical apoptotic change was demonstrated in RA synovial cells by each method. Anti-Fas antibody induced apoptotic synovial cell death in vitro.
CONCLUSION: This is the first reported study to demonstrate apoptosis in RA synovial cells. The findings indicate that rheumatoid synoviocytes undergo Fas-mediated apoptosis.

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Year:  1995        PMID: 7536416     DOI: 10.1002/art.1780380405

Source DB:  PubMed          Journal:  Arthritis Rheum        ISSN: 0004-3591


  60 in total

Review 1.  Synovial biopsy in arthritis research: five years of concerted European collaboration.

Authors:  B Bresnihan; P P Tak; P Emery; L Klareskog; F Breedveld
Journal:  Ann Rheum Dis       Date:  2000-07       Impact factor: 19.103

Review 2.  New therapies in development for autoimmune diseases: their rationale for combination treatment.

Authors:  V Strand
Journal:  Springer Semin Immunopathol       Date:  2001

3.  Amelioration of collagen-induced arthritis by CD95 (Apo-1/Fas)-ligand gene transfer.

Authors:  H Zhang; Y Yang; J L Horton; E B Samoilova; T A Judge; L A Turka; J M Wilson; Y Chen
Journal:  J Clin Invest       Date:  1997-10-15       Impact factor: 14.808

4.  Synoviolin/Hrd1, an E3 ubiquitin ligase, as a novel pathogenic factor for arthropathy.

Authors:  Tetsuya Amano; Satoshi Yamasaki; Naoko Yagishita; Kaneyuki Tsuchimochi; Hiroshi Shin; Ko-ichi Kawahara; Satoko Aratani; Hidetoshi Fujita; Lei Zhang; Rie Ikeda; Ryoji Fujii; Naoki Miura; Setsuro Komiya; Kusuki Nishioka; Ikuro Maruyama; Akiyoshi Fukamizu; Toshihiro Nakajima
Journal:  Genes Dev       Date:  2003-09-15       Impact factor: 11.361

5.  Chlorogenic acid induces apoptosis to inhibit inflammatory proliferation of IL-6-induced fibroblast-like synoviocytes through modulating the activation of JAK/STAT and NF-κB signaling pathways.

Authors:  Lixia Lou; Jingwei Zhou; Yujun Liu; Y I Wei; Jiuli Zhao; Jiagang Deng; Bin Dong; Lingqun Zhu; Aiming Wu; Yingxi Yang; Limin Chai
Journal:  Exp Ther Med       Date:  2016-03-09       Impact factor: 2.447

Review 6.  Molecular mechanism of immune response, synovial proliferation and apoptosis in rheumatoid arthritis.

Authors:  T Hasunuma; T Kato; T Kobata; K Nishioka
Journal:  Springer Semin Immunopathol       Date:  1998

7.  Apoptosis in rheumatoid arthritis: p53 overexpression in rheumatoid arthritis synovium.

Authors:  G S Firestein; K Nguyen; K R Aupperle; M Yeo; D L Boyle; N J Zvaifler
Journal:  Am J Pathol       Date:  1996-12       Impact factor: 4.307

8.  CIP2A facilitates apoptotic resistance of fibroblast-like synoviocytes in rheumatoid arthritis independent of c-Myc expression.

Authors:  Jaejoon Lee; Hyemin Jeong; Eun-Jung Park; Ji Won Hwang; Bo Huang; Eun-Kyung Bae; Joong Kyong Ahn; Hoon-Suk Cha; Eun-Mi Koh
Journal:  Rheumatol Int       Date:  2013-03-02       Impact factor: 2.631

9.  Ceramide, a mediator of interleukin 1, tumour necrosis factor alpha, as well as Fas receptor signalling, induces apoptosis of rheumatoid arthritis synovial cells.

Authors:  N Mizushima; H Kohsaka; N Miyasaka
Journal:  Ann Rheum Dis       Date:  1998-08       Impact factor: 19.103

10.  Akt activity protects rheumatoid synovial fibroblasts from Fas-induced apoptosis by inhibition of Bid cleavage.

Authors:  Samuel García; Myriam Liz; Juan J Gómez-Reino; Carmen Conde
Journal:  Arthritis Res Ther       Date:  2010-02-26       Impact factor: 5.156

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