Literature DB >> 7498663

Role of hepatocytes in direct clearance of lipopolysaccharide in rats.

Y Mimura1, S Sakisaka, M Harada, M Sata, K Tanikawa.   

Abstract

BACKGROUND & AIMS: The liver is the clearance organ for lipopolysaccharide (LPS). The aim of this study was to investigate the biliary excretion of LPS using fluorescein isothiocyanate (FITC)-labeled LPS.
METHODS: After FITC-LPS was injected intravenously into rats, the cellular localization of fluorescence in the liver was examined and the biliary excretion of fluorescence was measured. The effects of gadolinium chloride, a blocker of Kupffer cells, and colchicine, an inhibitor of microtubules, on the biliary excretion of fluorescence was investigated, and bile was analyzed using high-performance liquid chromatography.
RESULTS: Laser scanning confocal microscopy showed that fluorescence was taken up by hepatocytes 5 minutes after injection of FITC-LPS into the portal vein. When FITC-LPS was injected into the portal vein, fluorescence was rapidly secreted into bile, peaking at 20 minutes, and 25.1% of the injected dose appeared in bile within 60 minutes. When the same dose of FITC-LPS was injected into the tail vein, 15.8% appeared in bile within 60 minutes. Chromatography showed that FITC-LPS was excreted into bile in an unchanged form over a period of 20 minutes after injection. Colchicine significantly reduced the biliary excretion of fluorescence, but gadolinium chloride had no effect.
CONCLUSIONS: LPS was directly and effectively processed by hepatocytes and secreted into the bile canalicular system via a microtubule-dependent vesicular pathway.

Entities:  

Mesh:

Substances:

Year:  1995        PMID: 7498663     DOI: 10.1016/0016-5085(95)90765-3

Source DB:  PubMed          Journal:  Gastroenterology        ISSN: 0016-5085            Impact factor:   22.682


  58 in total

Review 1.  Primary biliary cirrhosis: From bench to bedside.

Authors:  Elias Kouroumalis; George Notas
Journal:  World J Gastrointest Pharmacol Ther       Date:  2015-08-06

Review 2.  The Riddle of Nonalcoholic Fatty Liver Disease: Progression From Nonalcoholic Fatty Liver to Nonalcoholic Steatohepatitis.

Authors:  Mithun Sharma; Shasikala Mitnala; Ravi K Vishnubhotla; Rathin Mukherjee; Duvvur N Reddy; Padaki N Rao
Journal:  J Clin Exp Hepatol       Date:  2015-02-16

3.  Endotoxin stimulates liver macrophages to release mediators that inhibit an early step in hepadnavirus replication.

Authors:  U Klöcker; U Schultz; H Schaller; U Protzer
Journal:  J Virol       Date:  2000-06       Impact factor: 5.103

4.  Loss of CFTR affects biliary epithelium innate immunity and causes TLR4-NF-κB-mediated inflammatory response in mice.

Authors:  Romina Fiorotto; Roberto Scirpo; Michael Trauner; Luca Fabris; Rafaz Hoque; Carlo Spirli; Mario Strazzabosco
Journal:  Gastroenterology       Date:  2011-06-26       Impact factor: 22.682

5.  Protective effect of the intravenous administration of ursodeoxycholic acid against endotoxemia in rats with obstructive jaundice.

Authors:  Y Hori; H Ohyanagi
Journal:  Surg Today       Date:  1997       Impact factor: 2.549

6.  ETS Proto-oncogene 1 Transcriptionally Up-regulates the Cholangiocyte Senescence-associated Protein Cyclin-dependent Kinase Inhibitor 2A.

Authors:  Steven P O'Hara; Patrick L Splinter; Christy E Trussoni; Maria J Lorenzo Pisarello; Lorena Loarca; Noah S Splinter; Bryce F Schutte; Nicholas F LaRusso
Journal:  J Biol Chem       Date:  2017-02-08       Impact factor: 5.157

7.  Hepatic uptake and deacylation of the LPS in bloodborne LPS-lipoprotein complexes.

Authors:  Baomei Shao; Robert S Munford; Richard Kitchens; Alan W Varley
Journal:  Innate Immun       Date:  2012-03-22       Impact factor: 2.680

8.  A simple mathematical model of signaling resulting from the binding of lipopolysaccharide with Toll-like receptor 4 demonstrates inherent preconditioning behavior.

Authors:  Beatrice Rivière; Yekaterina Epshteyn; David Swigon; Yoram Vodovotz
Journal:  Math Biosci       Date:  2008-10-11       Impact factor: 2.144

9.  Endotoxin uptake in mouse liver is blocked by endotoxin pretreatment through a suppressor of cytokine signaling-1-dependent mechanism.

Authors:  Melanie J Scott; Shubing Liu; Richard A Shapiro; Yoram Vodovotz; Timothy R Billiar
Journal:  Hepatology       Date:  2009-05       Impact factor: 17.425

Review 10.  Regulation of wound healing and organ fibrosis by toll-like receptors.

Authors:  Peter Huebener; Robert F Schwabe
Journal:  Biochim Biophys Acta       Date:  2012-12-04
View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.