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14 in total

1. Second window of preconditioning normalizes palmitate use for oxidation and improves function during low-flow ischaemia.

Authors: Raymond K Kudej; Mathew Fasano; Xin Zhao; Gary D Lopaschuk; Susan K Fischer; Dorothy E Vatner; Stephen F Vatner; E Douglas Lewandowski
Journal: Cardiovasc Res Date: 2011-08-11 Impact factor: 10.787

Review 2. Metabolic therapy for ischemic heart disease: the rationale for inhibition of fatty acid oxidation.

Authors: William C Stanley; Hani N Sabbah
Journal: Heart Fail Rev Date: 2005-12 Impact factor: 4.214

Review 3. Metabolic therapy for heart disease: impact of trimetazidine.

Authors: Hani N Sabbah; William C Stanley
Journal: Heart Fail Rev Date: 2005-12 Impact factor: 4.214

4. Effects of amiodarone on cardiac function and mitochondrial oxidative phosphorylation during ischemia and reperfusion.

Authors: D Moreau; F Clauw; L Martine; A Grynberg; L Rochette; L Demaison
Journal: Mol Cell Biochem Date: 1999-04 Impact factor: 3.396

5. Systemic response to thermal injury in rats. Accelerated protein degradation and altered glucose utilization in muscle.

Authors: A S Clark; R A Kelly; W E Mitch
Journal: J Clin Invest Date: 1984-09 Impact factor: 14.808

Oxfenicine diverts rat muscle metabolism from fatty acid to carbohydrate oxidation and protects the ischaemic rat heart.

1. Second window of preconditioning normalizes palmitate use for oxidation and improves function during low-flow ischaemia.

Review 2. Metabolic therapy for ischemic heart disease: the rationale for inhibition of fatty acid oxidation.

Review 3. Metabolic therapy for heart disease: impact of trimetazidine.

4. Effects of amiodarone on cardiac function and mitochondrial oxidative phosphorylation during ischemia and reperfusion.

5. Systemic response to thermal injury in rats. Accelerated protein degradation and altered glucose utilization in muscle.

Review 6. Metabolic approaches to the treatment of ischemic heart disease: the clinicians' perspective.

Review 7. Energy metabolism in the normal and failing heart: potential for therapeutic interventions.

8. Malonyl-CoA metabolism in cardiac myocytes and its relevance to the control of fatty acid oxidation.

9. Two mechanisms produce tissue-specific inhibition of fatty acid oxidation by oxfenicine.

10. The effect of Oxfenicine on cardiac carbohydrate metabolism in intact dogs.