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12 in total

1. Determination of lead in paired samples of human blood and synovial fluid.

Authors: A Villegas-Navarro; D Rosales; E Bustos; R Reyes; J L Reyes; T A Dieck; A Heredia
Journal: Bull Environ Contam Toxicol Date: 1992-09 Impact factor: 2.151

2. Measurement by ICP-MS of lead in plasma and whole blood of lead workers and controls.

Authors: A Schütz; I A Bergdahl; A Ekholm; S Skerfving
Journal: Occup Environ Med Date: 1996-11 Impact factor: 4.402

3. Biochemical diagnosis of occupational exposure to lead toxicity.

Authors: B V Somashekaraiah; B Venkaiah; A R Prasad
Journal: Bull Environ Contam Toxicol Date: 1990-02 Impact factor: 2.151

4. delta-Aminolevulinic acid dehydratase genotype modifies four hour urinary lead excretion after oral administration of dimercaptosuccinic acid.

Authors: B S Schwartz; B K Lee; W Stewart; P Sithisarankul; P T Strickland; K D Ahn; K Kelsey
Journal: Occup Environ Med Date: 1997-04 Impact factor: 4.402

5. Lead transport and binding by human erythrocytes in vitro.

Authors: T J Simons
Journal: Pflugers Arch Date: 1993-05 Impact factor: 3.657

6. The effect of low lead doses in vitro and in vivo on the d-ala-d activity of erythrocytes, bone marrow cells, liver and brain of the mouse.

Authors: E Schlick; K Mengel; K D Friedberg
Journal: Arch Toxicol Date: 1983-07 Impact factor: 5.153

Erythrocyte lead-binding protein after occupational exposure. I. Relationship to lead toxicity.

1. Determination of lead in paired samples of human blood and synovial fluid.

2. Measurement by ICP-MS of lead in plasma and whole blood of lead workers and controls.

3. Biochemical diagnosis of occupational exposure to lead toxicity.

4. delta-Aminolevulinic acid dehydratase genotype modifies four hour urinary lead excretion after oral administration of dimercaptosuccinic acid.

5. Lead transport and binding by human erythrocytes in vitro.

6. The effect of low lead doses in vitro and in vivo on the d-ala-d activity of erythrocytes, bone marrow cells, liver and brain of the mouse.

7. Mechanisms of ALA-D inhibition by lead and of its restoration by zinc and dithiothreitol.

Review 8. Chronic low-level lead exposure. Its role in the pathogenesis of hypertension.

9. Relationships between distribution of lead in erythrocytes in vivo and in vitro and inhibition of ALA-D.

10. Lead-binding proteins: a review.