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Literature DB >> 7155941

Patent ductus arteriosus in premature infants: a review of current management.

Abstract

Problems related to the ductus arteriosus confront the pediatrician more than any other isolated cardiac defect. Due to increased awareness of the problems of prematurity, the reported frequency of patent ductus arteriosus increased threefold between 1970 and 1975. Aggressive therapy directed toward closure of the ductus is indicated within the first week of life in the very immature infant. Pharmacologic closure of the PDA with indomethacin is effective about half the time.

Entities: Chemical Disease Species

Mesh：

Substances：
Indomethacin

Year: 1982 PMID： 7155941 DOI： 10.1007/BF02082334

Source DB: PubMed Journal: Pediatr Cardiol ISSN： 0172-0643 Impact factor: 1.655

36 in total

1. RESPONSE OF DUCTUS ARTERIOSUS AND PULMONARY AND SYSTEMIC ARTERIAL PRESSURE TO CHANGES IN OXYGEN ENVIRONMENT IN NEWBORN INFANTS.

Authors: A J MOSS; G C EMMANOUILIDES; F H ADAMS; K CHUANG
Journal: Pediatrics Date: 1964-06 Impact factor: 7.124

2. The structure of the pulmonary arterial tree in fetal and early postnatal life.

Authors: C A WAGENVOORT; H N NEUFELD; J E EDWARDS
Journal: Lab Invest Date: 1961 Jul-Aug Impact factor: 5.662

3. Failure of indomethacin therapy to induce closure of patent ductus arteriosus in premature infants with respiratory distress syndrome.

Authors: W A Neal; J M Kyle; M D Mullett
Journal: J Pediatr Date: 1977-10 Impact factor: 4.406

1. Introduction of plasma indomethacin level monitoring and evaluation of an effective threshold level in very low birth weight infants with symptomatic patent ductus arteriosus.

Authors: H W Seyberth; G Knapp; D Wolf; H E Ulmer
Journal: Eur J Pediatr Date: 1983-12 Impact factor: 3.183

1 in total

Patent ductus arteriosus in premature infants: a review of current management.

1. RESPONSE OF DUCTUS ARTERIOSUS AND PULMONARY AND SYSTEMIC ARTERIAL PRESSURE TO CHANGES IN OXYGEN ENVIRONMENT IN NEWBORN INFANTS.

2. The structure of the pulmonary arterial tree in fetal and early postnatal life.

3. Failure of indomethacin therapy to induce closure of patent ductus arteriosus in premature infants with respiratory distress syndrome.

4. Demonstration of adrenergic receptor function and innervation in the ductus arteriosus of the human fetus.

5. Poor response to oral indomethacin therapy for persistent ductus arteriosus in very low birthweight infants.

6. Patent ductus arteriosus complicating the respiratory distress syndrome in preterm infants.

7. Patent ductus arteriosus complicating respiratory distress syndrome.

8. Prostaglandin I2 is less relaxant than prostaglandin E2 on the lamb ductus arteriosus.

9. Ligation of patent ductus arteriosus in the very low birthweight newborn infant.

10. Indomethacin disposition and indomethacin-induced platelet dysfunction in premature infants.

1. Introduction of plasma indomethacin level monitoring and evaluation of an effective threshold level in very low birth weight infants with symptomatic patent ductus arteriosus.