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8 in total

1. Immunolocalization of hyperpolarization-activated cationic HCN1 and HCN3 channels in the rat nephron: regulation of HCN3 by potassium diets.

Authors: Zinaeli López-González; Cosete Ayala-Aguilera; Flavio Martinez-Morales; Othir Galicia-Cruz; Carolina Salvador-Hernández; José Pedraza-Chaverri; Mara Medeiros; Ana Maria Hernández; Laura I Escobar
Journal: Histochem Cell Biol Date: 2015-10-29 Impact factor: 4.304

2. Potassium depletion increases luminal Na+/H+ exchange and basolateral Na+:CO3=:HCO3- cotransport in rat renal cortex.

Authors: M Soleimani; J A Bergman; M A Hosford; T D McKinney
Journal: J Clin Invest Date: 1990-10 Impact factor: 14.808

3. Performance of Predictive Equations and Biochemical Measures Quantifying Net Endogenous Acid Production and the Potential Renal Acid Load.

Authors: Benjamin H Parmenter; Michael Dymock; Tanushree Banerjee; Anthony Sebastian; Gary J Slater; Lynda A Frassetto
Journal: Kidney Int Rep Date: 2020-07-29

4. Metabolic alkalosis in the rat. Evidence that reduced glomerular filtration rather than enhanced tubular bicarbonate reabsorption is responsible for maintaining the alkalotic state.

Authors: M G Cogan; F Y Liu
Journal: J Clin Invest Date: 1983-05 Impact factor: 14.808

8. The Cl^-/HCO₃^- exchanger pendrin is downregulated during oral co-administration of exogenous mineralocorticoid and KCl in patients with primary aldosteronism.

Authors: Aihua Wu; Martin J Wolley; Qi Wu; Richard D Gordon; Robert A Fenton; Michael Stowasser
Journal: J Hum Hypertens Date: 2020-11-10 Impact factor: 3.012

8 in total

Systemic and renal acid-base effects of chronic dietary potassium depletion in humans.

1. Immunolocalization of hyperpolarization-activated cationic HCN1 and HCN3 channels in the rat nephron: regulation of HCN3 by potassium diets.

2. Potassium depletion increases luminal Na+/H+ exchange and basolateral Na+:CO3=:HCO3- cotransport in rat renal cortex.

3. Performance of Predictive Equations and Biochemical Measures Quantifying Net Endogenous Acid Production and the Potential Renal Acid Load.

4. Metabolic alkalosis in the rat. Evidence that reduced glomerular filtration rather than enhanced tubular bicarbonate reabsorption is responsible for maintaining the alkalotic state.

5. Regulation of collecting tubule adenosine triphosphatases by aldosterone and potassium.

6. Treatment with potassium bicarbonate lowers calcium excretion and bone resorption in older men and women.

7. Evidence for ammonium conductance in a mouse thick ascending limb cell line.

8. The Cl^-/HCO₃^- exchanger pendrin is downregulated during oral co-administration of exogenous mineralocorticoid and KCl in patients with primary aldosteronism.