Literature DB >> 7064172

Pure motor hemiplegia: CT study of 30 cases.

A Rascol, M Clanet, C Manelfe, B Guiraud, A Bonafe.   

Abstract

Pure motor hemiplegia (PMH) is a well defined syndrome usually caused by ischemic lesions of lacunar type located either in the internal capsule or in the pons. Angiography and isotope scanning are usually normal. CT scan reveals small deep infarcts and appears to be the most reliable investigative method. The CT scan findings are described of thirty patients with PMH of rapid onset (less than 36 hours). In 29 of the 30 cases a lesion was found which could explain the PMH. Small hemorrhages (2 cm in diameter) in the posterior limb of the internal capsule were noted in two cases. Ischemic lesions were found in 27 patients, 22 patients had a single lesion (20 capsular and 2 pontine), while 5 patients had 2 lesions (2 bi-capsular, 3 capsular and pontine). Three varieties of ischemic capsular lesions were observed. We found in 15 cases a capsulo-putamine-caudate infarct (type I); in 8 cases a capsulo-pallidal infarct (type II); and in 2 cases an anterior capsulo-caudate infarct (type III). Type I corresponded to the area of the lateral lenticulostriate branches of the middle cerebral artery. Type II involved the territory of the perforating branches of the anterior choroidal artery. We suggest that type III involves the territory of the internal lenticulostriate branches of the anterior cerebral artery. Lacunes are generally linked to arterial systemic hypertension. However, only 16 of 30 patients in this series were chronically hypertensive.

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Year:  1982        PMID: 7064172     DOI: 10.1161/01.str.13.1.11

Source DB:  PubMed          Journal:  Stroke        ISSN: 0039-2499            Impact factor:   7.914


  15 in total

1.  Anatomical bases for surgical approach to the initial segment of the anterior cerebral artery. Microanatomy of Heubner's artery and perforating branches of the anterior cerebral artery.

Authors:  S Marinković; M Milisavljević; M Kovacević
Journal:  Surg Radiol Anat       Date:  1986       Impact factor: 1.246

2.  Non ischaemic causes of lacunar syndromes: prevalence and clinical findings.

Authors:  N Anzalone; G Landi
Journal:  J Neurol Neurosurg Psychiatry       Date:  1989-10       Impact factor: 10.154

Review 3.  Current management of ischaemic stroke.

Authors:  R S Marshall; J P Mohr
Journal:  J Neurol Neurosurg Psychiatry       Date:  1993-01       Impact factor: 10.154

4.  Carotid artery bruits and lacunar strokes.

Authors:  D A Hindson
Journal:  West J Med       Date:  1984-05

5.  X-ray computed tomography (CT) study of small, deep and recent infarcts (SDRIs) of the cerebral hemispheres in adults. Preliminary and critical report.

Authors:  M Launay; M N'Diaye; J Bories
Journal:  Neuroradiology       Date:  1985       Impact factor: 2.804

6.  Acute capsular infarction. Location of the lesions and the clinical features.

Authors:  M Kashihara; K Matsumoto
Journal:  Neuroradiology       Date:  1985       Impact factor: 2.804

7.  Lacunar syndromes due to brainstem infarct and haemorrhage.

Authors:  C Huang; E Woo; Y L Yu; F L Chan
Journal:  J Neurol Neurosurg Psychiatry       Date:  1988-04       Impact factor: 10.154

8.  Role of computed tomography in vertebrobasilar ischemia.

Authors:  A Bonafe; C Manelfe; B Scotto; M Y Pradere; A Rascol
Journal:  Neuroradiology       Date:  1985       Impact factor: 2.804

9.  Diffusion tensor pyramidal tractography in patients with anterior choroidal artery infarcts.

Authors:  M Nelles; J Gieseke; S Flacke; L Lachenmayer; H H Schild; H Urbach
Journal:  AJNR Am J Neuroradiol       Date:  2007-12-13       Impact factor: 3.825

10.  An evaluation of cognitive disorders after anterior choroidal artery infarction.

Authors:  Marc Rousseaux; Maryline Cabaret; Rawabi Serafi; Odile Kozlowski
Journal:  J Neurol       Date:  2008-06-30       Impact factor: 4.849

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