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Literature DB >> 7018975

Nesidioblastosis of the pancreas in infancy.

A Aynsley-Green.

Abstract

Entities: Disease

Mesh：

Substances：
Insulin

Year: 1981 PMID： 7018975

Source DB: PubMed Journal: Dev Med Child Neurol ISSN： 0012-1622 Impact factor: 5.449

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Cited

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11 in total

1. Dominantly inherited hyperinsulinism caused by a mutation in the sulfonylurea receptor type 1.

Authors: H Huopio; F Reimann; R Ashfield; J Komulainen; H L Lenko; J Rahier; I Vauhkonen; J Kere; M Laakso; F Ashcroft; T Otonkoski
Journal: J Clin Invest Date: 2000-10 Impact factor: 14.808

2. Somatic deletion of the imprinted 11p15 region in sporadic persistent hyperinsulinemic hypoglycemia of infancy is specific of focal adenomatous hyperplasia and endorses partial pancreatectomy.

Authors: P de Lonlay; J C Fournet; J Rahier; M S Gross-Morand; F Poggi-Travert; V Foussier; J P Bonnefont; M C Brusset; F Brunelle; J J Robert; C Nihoul-Fékété; J M Saudubray; C Junien
Journal: J Clin Invest Date: 1997-08-15 Impact factor: 14.808

3. Therapy for persistent hyperinsulinemic hypoglycemia of infancy. Understanding the responsiveness of beta cells to diazoxide and somatostatin.

Authors: C Kane; K J Lindley; P R Johnson; R F James; P J Milla; A Aynsley-Green; M J Dunne
Journal: J Clin Invest Date: 1997-10-01 Impact factor: 14.808

Nesidioblastosis of the pancreas in infancy.

1. Dominantly inherited hyperinsulinism caused by a mutation in the sulfonylurea receptor type 1.

2. Somatic deletion of the imprinted 11p15 region in sporadic persistent hyperinsulinemic hypoglycemia of infancy is specific of focal adenomatous hyperplasia and endorses partial pancreatectomy.

3. Therapy for persistent hyperinsulinemic hypoglycemia of infancy. Understanding the responsiveness of beta cells to diazoxide and somatostatin.

4. Lessons in human biology from a monogenic pancreatic β cell disease.

5. Hyperinsulinism in mice with heterozygous loss of K(ATP) channels.

6. Targeting the cell cycle inhibitor p57Kip2 promotes adult human β cell replication.

7. Surgical treatment of hyperinsulinaemic hypoglycaemia in infancy and childhood.

8. Experience with 95% pancreatectomy and splenic salvage for neonatal nesidioblastosis.

Review 9. Hyperinsulinism and diabetes: genetic dissection of beta cell metabolism-excitation coupling in mice.

Review 10. Role of ZAC1 in transient neonatal diabetes mellitus and glucose metabolism.