Literature DB >> 7010877

Experimental transmission of human subacute spongiform encephalopathy to small rodents. III. Further transmission from three patients and distribution patterns of lesions in mice.

J Tateishi, H Doi, Y Sato, M Suetsugu, K Ishii, Y Kuroiwa.   

Abstract

Further experimental transmission of Creutzfeldt-Jakob disease (CJD) from three patients to mice and rats was carried out successfully. The clinical signs and pathologic features of spongiform encephalopathy transmitted to animals were much the same as in previous experiments, except that distribution of the lesions in the mice differed with each inoculated material taken from the patients. These observations suggest that multiplicity of CJD agents, as in the case of scrapie agents.

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Year:  1981        PMID: 7010877     DOI: 10.1007/bf00689997

Source DB:  PubMed          Journal:  Acta Neuropathol        ISSN: 0001-6322            Impact factor:   17.088


  2 in total

1.  Scrapie in mice. Agent-strain differences in the distribution and intensity of grey matter vacuolation.

Authors:  H Fraser; A G Dickinson
Journal:  J Comp Pathol       Date:  1973-01       Impact factor: 1.311

2.  Experimental transmission of human subacute spongiform encephalopathy to small rodents. I. Clinical and histological observations.

Authors:  J Tateishi; Y Sato; M Koga; H Doi; M Ohta
Journal:  Acta Neuropathol       Date:  1980       Impact factor: 17.088

  2 in total
  8 in total

1.  Abnormal isoform of prion protein accumulates in follicular dendritic cells in mice with Creutzfeldt-Jakob disease.

Authors:  T Kitamoto; T Muramoto; S Mohri; K Doh-Ura; J Tateishi
Journal:  J Virol       Date:  1991-11       Impact factor: 5.103

2.  Accumulation of abnormal prion protein in mice infected with Creutzfeldt-Jakob disease via intraperitoneal route: a sequential study.

Authors:  T Muramoto; T Kitamoto; J Tateishi; I Goto
Journal:  Am J Pathol       Date:  1993-11       Impact factor: 4.307

3.  Species barrier prevents an abnormal isoform of prion protein from accumulating in follicular dendritic cells of mice with Creutzfeldt-Jakob disease.

Authors:  T Muramoto; T Kitamoto; M Z Hoque; J Tateishi; I Goto
Journal:  J Virol       Date:  1993-11       Impact factor: 5.103

4.  The sequential development of abnormal prion protein accumulation in mice with Creutzfeldt-Jakob disease.

Authors:  T Muramoto; T Kitamoto; J Tateishi; I Goto
Journal:  Am J Pathol       Date:  1992-06       Impact factor: 4.307

5.  Antibiotics and antivirals do not modify experimentally-induced Creutzfeldt-Jakob disease in mice.

Authors:  J Tateishi
Journal:  J Neurol Neurosurg Psychiatry       Date:  1981-08       Impact factor: 10.154

6.  Immunohistochemical confirmation of Creutzfeldt-Jakob disease with a long clinical course with amyloid plaque core antibodies.

Authors:  T Kitamoto; J Tateishi
Journal:  Am J Pathol       Date:  1988-06       Impact factor: 4.307

7.  Early MRI findings in Creutzfeldt-Jakob disease.

Authors:  M Onofrj; T Fulgente; D Gambi; G Macchi
Journal:  J Neurol       Date:  1993-07       Impact factor: 4.849

8.  Evidence for an unconventional virus in mouse-adapted Creutzfeldt-Jakob disease.

Authors:  D T Kingsbury; D A Smeltzer; H L Amyx; C J Gibbs; D C Gajdusek
Journal:  Infect Immun       Date:  1982-09       Impact factor: 3.441

  8 in total

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