Literature DB >> 6637419

Amaurosis fugax. A unselected material.

P N Sørensen.   

Abstract

Forty-four unselected patients with amaurosis fugax (AF) have been followed for 4.6 years (mean 2.6). Thirty per cent had atheromatous lesions, 20% had miscellaneous diagnoses (temporal arteritis 5, transitory ocular hypertension 2, glaucomatous iritis 1, benign intracraniel hypertension 1), 50% consisted of young, mainly women, in whom no cause was found. Prior to AF 2 had hemiplegia, 4 TCI, one optic atrophy and suspicion in 3. None died, one developed hemiparesis and one macular degeneration. An individual conservative attitude to AF seems justified in this material.

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Year:  1983        PMID: 6637419     DOI: 10.1111/j.1755-3768.1983.tb04348.x

Source DB:  PubMed          Journal:  Acta Ophthalmol (Copenh)        ISSN: 0001-639X


  3 in total

1.  Protein C deficiency: a cause of amaurosis fugax?

Authors:  D B Smith; G E Ens
Journal:  J Neurol Neurosurg Psychiatry       Date:  1987-03       Impact factor: 10.154

2.  The source of embolism in amaurosis fugax and retinal artery occlusion.

Authors:  R L Smit; G S Baarsma; P J Koudstaal
Journal:  Int Ophthalmol       Date:  1994       Impact factor: 2.031

3.  Internal jugular venous abnormalities in transient monocular blindness.

Authors:  Chun-Yu Cheng; Feng-Chi Chang; A-Ching Chao; Chih-Ping Chung; Han-Hwa Hu
Journal:  BMC Neurol       Date:  2013-07-22       Impact factor: 2.474

  3 in total

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