Literature DB >> 656991

[Critical study of the hemodynamic status during anesthesia].

P Stieglitz, P Girardet.   

Abstract

Physicians must choose the anaesthetics for their patients and select the methods to cheek their haemodynamic status. Experimental works do not always bring sufficient information to help them in their daily practice. Circulatory reaction to a pharmacodynamic agent is diffuse and non-specific. Some examples drawn from theoretical considerations and practical situations support this view. The difficulty of assessing the actual damage caused by one apparent variation still remains. On the other hand, haemodynamic indices such as PA, CVP, dP/dt are composed of elementary data that are interdependent; so indices are interdependent, too. A puzzling fact is that depressing anaesthetics can initiate severe haemodynamic crisis, generally badly tolerated by tissues, but better tolerated by myocardium which is not definietely altered if the anoxia inflicted is accompanied by a real decrease in cardiac work. This explains the often slight effect of short anesthetic overdosage. Previous haemodynamic variations to be countered relative to anaesthesia are not really known. Biochemistry of the venous coronary blood does not yet bring striking features in this field.

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Year:  1978        PMID: 656991     DOI: 10.1007/BF03004878

Source DB:  PubMed          Journal:  Can Anaesth Soc J        ISSN: 0008-2856


  38 in total

1.  Hemodynamic determinants of oxygen consumption of the heart with special reference to the tension-time index.

Authors:  S J Sarnoff; E Braunwald; G H Welch; R B Case; W N Stainsby; R Macruz
Journal:  Am J Physiol       Date:  1958-01

2.  Comparison of isometric exercise and angiotensin infusion as stress test for evaluation of left ventricular function.

Authors:  R M Payne; L D Horwitz; C B Mullins
Journal:  Am J Cardiol       Date:  1973-04       Impact factor: 2.778

3.  Effects of blood volume expansion on left ventricular hemodynamics in man.

Authors:  V R Sanghvi; F Khaja; A L Mark; J O Parker
Journal:  Circulation       Date:  1972-10       Impact factor: 29.690

4.  Hemodynamic spectrum of myocardial infarction and cardiogenic shock. A conceptual model.

Authors:  H J Swan; J S Forrester; G Diamond; K Chatterjee; W W Parmley
Journal:  Circulation       Date:  1972-05       Impact factor: 29.690

5.  Transoesophageal aortic velography in man.

Authors:  P J Tomlin; F A Duck
Journal:  Can Anaesth Soc J       Date:  1975-09

6.  Alterations of normal left ventricular performance by general anesthesia.

Authors:  B E Filner; J S Karliner
Journal:  Anesthesiology       Date:  1976-12       Impact factor: 7.892

7.  Haemodynamic interactions of high-dose propranolol pretreatment and anaesthesia in the dog. II: The effects of acute arterial hypoxaemia at increasing depths of halothane anaesthesia.

Authors:  J G Roberts; P Foëx; T N Clarke; C Prys-Roberts; M J Bennett
Journal:  Br J Anaesth       Date:  1976-05       Impact factor: 9.166

8.  Haemodynamic interactions of high-dose propranolol pretreatment and anaesthesia in the dog. III: The effects of haemorrhage during halothane and trichloroethylene anaesthesia.

Authors:  J G Roberts; P Foëx; T N Clarke; M J Bennett; C A Saner
Journal:  Br J Anaesth       Date:  1976-05       Impact factor: 9.166

9.  Effects of ketamine on canine cardiovascular function.

Authors:  F A Diaz; J A Bianco; A Bello; N Beer; H Velarde; J P Izquierdo; R Jaen
Journal:  Br J Anaesth       Date:  1976-10       Impact factor: 9.166

10.  Control of myocardial oxygen consumption: relative influence of contractile state and tension development.

Authors:  T P Graham; J W Covell; E H Sonnenblick; J Ross; E Braunwald
Journal:  J Clin Invest       Date:  1968-02       Impact factor: 14.808

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