Literature DB >> 6476556

Brain injury by ischemic anoxia: hypothesis extension--a tale of two ions?

B C White, S D Aust, K E Arfors, L D Aronson.   

Abstract

The understanding of the pathophysiology of cerebral ischemic anoxia depends on elucidation of cellular neuronal changes during this event and under conditions of cardiopulmonary resuscitation. Current knowledge of these metabolic developments is reviewed, and a hypothesis of the role of iron in this situation is presented.

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Year:  1984        PMID: 6476556     DOI: 10.1016/s0196-0644(84)80461-8

Source DB:  PubMed          Journal:  Ann Emerg Med        ISSN: 0196-0644            Impact factor:   5.721


  4 in total

1.  Picroliv -- a natural product protects cells and regulates the gene expression during hypoxia/reoxygenation.

Authors:  J P Gaddipati; S Madhavan; G S Sidhu; A K Singh; P Seth; R K Maheshwari
Journal:  Mol Cell Biochem       Date:  1999-04       Impact factor: 3.396

2.  Iron deposition after transient forebrain ischemia in rat brain.

Authors:  Viera Danielisová; Miroslav Gottlieb; Jozef Burda
Journal:  Neurochem Res       Date:  2002-03       Impact factor: 3.996

Review 3.  Glucocorticoids as an emerging pharmacologic agent for cardiopulmonary resuscitation.

Authors:  Giolanda Varvarousi; Antonia Stefaniotou; Dimitrios Varvaroussis; Theodoros Xanthos
Journal:  Cardiovasc Drugs Ther       Date:  2014-10       Impact factor: 3.727

4.  Prognostic factors for neurological outcomes in Korean targeted temperature management recipients with return of spontaneous circulation after out-of-hospital cardiac arrests: A nationwide observational study.

Authors:  Jae Guk Kim; Hyungoo Shin; Hyun Young Choi; Wonhee Kim; Jihoon Kim; Shinje Moon; Bongyoung Kim; Chiwon Ahn; Juncheol Lee
Journal:  Medicine (Baltimore)       Date:  2020-04       Impact factor: 1.817

  4 in total

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