Literature DB >> 6371387

The ethanol withdrawal syndrome: a consequence of lack of substrate for a cerebral Krebs-cycle.

R F Derr.   

Abstract

The hypothesis is advanced that the ethanol withdrawal syndrome is a manifestation of dependence by a cerebral Krebs-cycle on substrates which are readily available during ethanol addiction but which are relatively unavailable during withdrawal.

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Year:  1984        PMID： 6371387     DOI： 10.1016/0022-5193(84)90036-5

Source DB:  PubMed          Journal:  J Theor Biol        ISSN： 0022-5193            Impact factor:   2.691


  2 in total

1.  Alcohol decreases baseline brain glucose metabolism more in heavy drinkers than controls but has no effect on stimulation-induced metabolic increases.

Authors:  Nora D Volkow; Gene-Jack Wang; Ehsan Shokri Kojori; Joanna S Fowler; Helene Benveniste; Dardo Tomasi
Journal:  J Neurosci       Date:  2015-02-18       Impact factor: 6.167

2.  Acute alcohol intoxication decreases glucose metabolism but increases acetate uptake in the human brain.

Authors:  Nora D Volkow; Sung Won Kim; Gene-Jack Wang; David Alexoff; Jean Logan; Lisa Muench; Colleen Shea; Frank Telang; Joanna S Fowler; Christopher Wong; Helene Benveniste; Dardo Tomasi
Journal:  Neuroimage       Date:  2012-08-28       Impact factor: 6.556
  2 in total

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