Literature DB >> 6351708

[Copper in ruminants. Review].

N Auza.   

Abstract

In ruminants, copper is absorbed in the first parts of the intestine. Mainly transported by ceruloplasmin, it concentrates in the liver; then it is eliminated by feces, bile, urine and milk. Copper intoxications are relatively rare. On the contrary, deficiencies are more frequent: they are due to a shortage of alimentary supply or to a competition with molybdenum and/or sulphate. These deficiencies induce the inhibition of copper-enzymes determining mainly troubles of hair pigmentation, reproduction and hematopoiesis. These troubles can be prevented or cured by the administration of copper salts.

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Year:  1983        PMID: 6351708

Source DB:  PubMed          Journal:  Ann Rech Vet        ISSN: 0003-4193


  4 in total

1.  Pathogenesis of neurological signs associated with bovine enteric coccidiosis: a prospective study and review.

Authors:  C M Isler; J E Bellamy; G A Wobeser
Journal:  Can J Vet Res       Date:  1987-04       Impact factor: 1.310

2.  Copper poisoning in a flock of sheep. Copper excretion patterns after treatment with molybdenum and sulfur or penicillamine.

Authors:  M Hidiroglou; D P Heaney; K E Hartin
Journal:  Can Vet J       Date:  1984-10       Impact factor: 1.008

3.  Copper poisoning in a dairy herd fed a mineral supplement.

Authors:  C H Bradley
Journal:  Can Vet J       Date:  1993-05       Impact factor: 1.008

4.  Animal models of copper-associated liver disease.

Authors:  I Carmen Fuentealba; Enrique M Aburto
Journal:  Comp Hepatol       Date:  2003-04-03
  4 in total

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