Literature DB >> 6333858

Acute abdominal emergencies during the first four weeks after spinal cord injury.

M H Berlly, C B Wilmot.   

Abstract

Diagnosing acute abdominal emergencies during spinal shock can be extremely difficult. Generally, the abdominal examination of an acutely cord-injured patient will not change with abdominal pathology. Loss of sensory, motor, and reflex functions mask typical signs. Nine hundred and forty-five medical charts were retrospectively examined to determine the incidence, causes, and risk factors for acute abdominal pathology during the first four weeks after spinal cord injury. Time of greatest risk was also investigated. Intraabdominal pathology was seen in 4.7% of the cases. Patients with complete cord lesions above the T5 level were most at risk. Respiratory distress was an additional risk factor for peptic ulcer disease. Decadron was found to be associated with pancreatitis but not with ulcers. Both pancreatitis and upper gastrointestinal hemorrhage appeared as early as three days postinjury. Findings of this study, as well as an understanding of the pathophysiology, symptomatology, and proper diagnostic workup assist in making the critical diagnosis.

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Year:  1984        PMID: 6333858

Source DB:  PubMed          Journal:  Arch Phys Med Rehabil        ISSN: 0003-9993            Impact factor:   3.966


  9 in total

1.  Time-course of recovery of gastric emptying and motility in rats with experimental spinal cord injury.

Authors:  E Qualls-Creekmore; M Tong; G M Holmes
Journal:  Neurogastroenterol Motil       Date:  2009-06-30       Impact factor: 3.598

2.  Diminished gastric prokinetic response to ghrelin in a rat model of spinal cord injury.

Authors:  E M Besecker; A R White; G M Holmes
Journal:  Neurogastroenterol Motil       Date:  2017-12-05       Impact factor: 3.598

3.  Gastric vagal afferent neuropathy following experimental spinal cord injury.

Authors:  Emily M Besecker; Emily N Blanke; Gina M Deiter; Gregory M Holmes
Journal:  Exp Neurol       Date:  2019-11-05       Impact factor: 5.330

4.  Mesenteric vascular dysregulation and intestinal inflammation accompanies experimental spinal cord injury.

Authors:  Emily M Besecker; Gina M Deiter; Nicole Pironi; Timothy K Cooper; Gregory M Holmes
Journal:  Am J Physiol Regul Integr Comp Physiol       Date:  2016-11-09       Impact factor: 3.619

5.  Experimental spinal cord injury in rats diminishes vagally-mediated gastric responses to cholecystokinin-8s.

Authors:  M Tong; E Qualls-Creekmore; K N Browning; R A Travagli; G M Holmes
Journal:  Neurogastroenterol Motil       Date:  2010-10-18       Impact factor: 3.598

6.  Spinal cord injury-mediated changes in electrophysiological properties of rat gastric nodose ganglion neurons.

Authors:  Emily N Blanke; Victor Ruiz-Velasco; Gregory M Holmes
Journal:  Exp Neurol       Date:  2021-11-16       Impact factor: 5.330

7.  Gastric dysreflexia after acute experimental spinal cord injury in rats.

Authors:  M Tong; G M Holmes
Journal:  Neurogastroenterol Motil       Date:  2008-12-19       Impact factor: 3.598

8.  Upper gastrointestinal dysmotility after spinal cord injury: is diminished vagal sensory processing one culprit?

Authors:  Gregory M Holmes
Journal:  Front Physiol       Date:  2012-07-17       Impact factor: 4.566

9.  JPN Guidelines for the management of acute pancreatitis: epidemiology, etiology, natural history, and outcome predictors in acute pancreatitis.

Authors:  Miho Sekimoto; Tadahiro Takada; Yoshifumi Kawarada; Koichi Hirata; Toshihiko Mayumi; Masahiro Yoshida; Masahiko Hirota; Yasutoshi Kimura; Kazunori Takeda; Shuji Isaji; Masaru Koizumi; Makoto Otsuki; Seiki Matsuno
Journal:  J Hepatobiliary Pancreat Surg       Date:  2006
  9 in total

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