Literature DB >> 6297896

Free N-acetylneuraminic acid in tissues in Salla disease and the enzymes involved in its metabolism.

M Renlund, M A Chester, A Lundblad, J Parkkinen, T Krusius.   

Abstract

Salla disease is a lysosomal storage disorder of unknown etiology, characterized biochemically by increased urinary excretion of N-acetylneuraminic acid. This compound has now been shown to occur in abnormally large amounts in liver and cultured skin fibroblasts from these patients. Quantification of N-acetylneuraminic acid was performed using a new gas-chromatography/mass spectrometric single-ion method which is sensitive and specific. No abnormalities in the activity of several enzymes involved in sialic acid metabolism (N-acetylneuraminate:pyruvate lyase, neuraminidase, CMP-N-acetylneuraminate N-acylneuraminohydrolase and CTP:N-acyl-neuraminate cytidylyltransferase) were demonstrable. A possible explanation for the defect is a malfunctioning active transport of N-acetylneuraminic acid across the lysosomal membrane.

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Year:  1983        PMID: 6297896     DOI: 10.1111/j.1432-1033.1983.tb07114.x

Source DB:  PubMed          Journal:  Eur J Biochem        ISSN: 0014-2956


  10 in total

1.  Storage material from urine and tissues in the nephropathic phenotype of infantile sialic acid storage disease.

Authors:  E Paschke; W Gruber; E Ring; W Sperl
Journal:  J Inherit Metab Dis       Date:  1992       Impact factor: 4.982

2.  Free N-acetylneuraminic acid (NANA) storage disorders: evidence for defective NANA transport across the lysosomal membrane.

Authors:  G M Mancini; F W Verheijen; H Galjaard
Journal:  Hum Genet       Date:  1986-07       Impact factor: 4.132

3.  The effect of D-(+)-glucosamine on levels of free N-acetylneuraminic acid and UDP-N-acetylhexosamines in infantile sialic acid storage disease (ISSD) fibroblasts.

Authors:  E Paschke; G Höfler; A Roscher
Journal:  J Inherit Metab Dis       Date:  1987       Impact factor: 4.982

4.  N-Acetylneuraminic acid storage disease.

Authors:  J Baumkötter; M Cantz; K Mendla; W Baumann; H Friebolin; J Gehler; J Spranger
Journal:  Hum Genet       Date:  1985       Impact factor: 4.132

5.  The sialic acid residue of exogenous GM1 ganglioside is recycled for biosynthesis of sialoglycoconjugates in rat liver.

Authors:  R Ghidoni; M Trinchera; S Sonnino; V Chigorno; G Tettamanti
Journal:  Biochem J       Date:  1987-10-01       Impact factor: 3.857

6.  Studies on the defect underlying the lysosomal storage of sialic acid in Salla disease. Lysosomal accumulation of sialic acid formed from N-acetyl-mannosamine or derived from low density lipoprotein in cultured mutant fibroblasts.

Authors:  M Renlund; P T Kovanen; K O Raivio; P Aula; C G Gahmberg; C Ehnholm
Journal:  J Clin Invest       Date:  1986-02       Impact factor: 14.808

7.  Free sialic acid storage disease. A new Italian case.

Authors:  A Fois; P Balestri; M A Farnetani; G M Mancini; P Borgogni; M A Margollicci; M Molinelli; C Alessandrini; R Gerli
Journal:  Eur J Pediatr       Date:  1987-03       Impact factor: 3.183

8.  Salla disease in one non-Finnish patient.

Authors:  B Echenne; M Vidal; I Maire; J C Michalski; P Baldet; J Astruc
Journal:  Eur J Pediatr       Date:  1986-09       Impact factor: 3.183

9.  Defective lysosomal release of glycoprotein-derived sialic acid in fibroblasts from patients with sialic acid storage disease.

Authors:  K Mendla; J Baumkötter; C Rosenau; B Ulrich-Bott; M Cantz
Journal:  Biochem J       Date:  1988-02-15       Impact factor: 3.857

Review 10.  Free sialic acid storage disorder: Progress and promise.

Authors:  Marjan Huizing; Mary E Hackbarth; David R Adams; Melissa Wasserstein; Marc C Patterson; Steven U Walkley; William A Gahl
Journal:  Neurosci Lett       Date:  2021-04-20       Impact factor: 3.046

  10 in total

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