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Literature DB >> 62828

Erythrocyte ghosts (Na+ + K+) ATPase activity in Duchenne's dystrophy and myotonia.

Abstract

In Duchenne muscular dystrophy the activity of (Na+ + K+)ATPase in erythrocyte ghosts is reduced and its reaction to ouabain is paradoxical both in low sodium and high sodium systems. No such changes were seen in a case of Becker dystrophy, in limb-girdle dystrophy, and in neurogenic atrophy of muscles. In myotonic dystrophy and congenital myotonia the activity of ATPase and its inhibition by ouabain were depressed.

Entities: Chemical Disease Gene

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Year: 1976 PMID： 62828 DOI： 10.1007/BF00313489

Source DB: PubMed Journal: J Neurol ISSN： 0340-5354 Impact factor: 4.849

57 in total

1. Abnormalities in membrane microviscosity and ion transport in genetic muscular dystrophy.

Authors: R I Sha'afi; S B Rodan; R L Hintz; S M Fernandez; G A Rodan
Journal: Nature Date: 1975-04-10 Impact factor: 49.962

2. Lipoprotein nature of red cell adenosine triphosphatase.

Authors: H J SCHATZMANN
Journal: Nature Date: 1962-11-17 Impact factor: 49.962

3. Electron spin resonance studies of erythrocytes from patients with myotonic muscular dystrophy.

Authors: D A Butterfield; D B Chesnut; A D Roses; S H Appel
Journal: Proc Natl Acad Sci U S A Date: 1974-03 Impact factor: 11.205

4. The effect of phospholipids on the apparent activation energy of (Na+-K+)-ATPase.

Authors: K Taniguchi; S Iida
Journal: Biochim Biophys Acta Date: 1972-08-09

Review 5. Membrane phenomena.

Authors: A Rothstein
Journal: Annu Rev Physiol Date: 1968 Impact factor: 19.318

6. Myotonia induced by diazacholesterol: increased (Na+ + K+)-ATPase activity of erythrocyte ghosts and development of cataracts.

Authors: J B Peter; R M Andiman; R L Bowman; T Nagatomo
Journal: Exp Neurol Date: 1973-12 Impact factor: 5.330

3. The sodium pump of erythrocytes from patients with Duchenne muscular dystrophy: effect of ouabain on the active sodium flux and on (Na+, K+)ATPase.

Authors: G Souweine; J C Bernard; Y Lasne; J Lachanat
Journal: J Neurol Date: 1978-03-09 Impact factor: 4.849

3 in total

Erythrocyte ghosts (Na+ + K+) ATPase activity in Duchenne's dystrophy and myotonia.

1. Abnormalities in membrane microviscosity and ion transport in genetic muscular dystrophy.

2. Lipoprotein nature of red cell adenosine triphosphatase.

3. Electron spin resonance studies of erythrocytes from patients with myotonic muscular dystrophy.

4. The effect of phospholipids on the apparent activation energy of (Na+-K+)-ATPase.

Review 5. Membrane phenomena.

6. Myotonia induced by diazacholesterol: increased (Na+ + K+)-ATPase activity of erythrocyte ghosts and development of cataracts.

7. Effects of cations on ouabain binding by intact human erythrocytes.

8. Protein kinase activity in erythrocyte ghosts of patients with myotonic muscular dystrophy.

9. Erythrocyte abnormality in human myopathy.

10. Sodium, potassium, and chloride fluxes in intercostal muscle from normal goats and goats with hereditary myotonia.

Review 1. Duchenne muscular dystrophy: pathogenetic aspects and genetic prevention.

2. Erythrocyte ghost (Na+ + K+) ATPase activity in mice with hereditary muscular dystrophy (strain C57 BL/64J/dy).

3. The sodium pump of erythrocytes from patients with Duchenne muscular dystrophy: effect of ouabain on the active sodium flux and on (Na+, K+)ATPase.