Literature DB >> 6259835

Cell killing by simian virus 40: the sequence of ultrastructural alterations leading to cellular degeneration and death.

K H Eggleton, L C Norkin.   

Abstract

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Year:  1981        PMID： 6259835     DOI： 10.1016/0042-6822(81)90009-x

Source DB:  PubMed          Journal:  Virology        ISSN： 0042-6822            Impact factor:   3.616


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  5 in total

1.  Simian virus 40 late proteins possess lytic properties that render them capable of permeabilizing cellular membranes.

Authors:  Robert Daniels; Nasser M Rusan; Anne-Kathrin Wilbuer; Leonard C Norkin; Patricia Wadsworth; Daniel N Hebert
Journal:  J Virol       Date:  2006-07       Impact factor: 5.103

Review 2.  Papovaviral persistent infections.

Authors:  L C Norkin
Journal:  Microbiol Rev       Date:  1982-12

Review 3.  Altered cellular morphology resulting from cytocidal virus infection.

Authors:  M Schrom; R Bablanian
Journal:  Arch Virol       Date:  1981       Impact factor: 2.574

4.  The abundant nuclear enzyme PARP participates in the life cycle of simian virus 40 and is stimulated by minor capsid protein VP3.

Authors:  Ariela Gordon-Shaag; Yael Yosef; Mahmoud Abd El-Latif; Ariella Oppenheim
Journal:  J Virol       Date:  2003-04       Impact factor: 5.103

Review 5.  Modification of membrane permeability by animal viruses.

Authors:  L Carrasco
Journal:  Adv Virus Res       Date:  1995       Impact factor: 9.937
  5 in total

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