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Literature DB >> 6241232

Phenotypic studies of cells from periodontal disease tissues.

M A Taubman, E D Stoufi, J L Ebersole, D J Smith.

Abstract

Mesh：

Year: 1984 PMID： 6241232 DOI： 10.1111/j.1600-0765.1984.tb01320.x

Source DB: PubMed Journal: J Periodontal Res ISSN： 0022-3484 Impact factor: 4.419

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10 in total

1. In vitro induction of activation-induced cell death in lymphocytes from chronic periodontal lesions by exogenous Fas ligand.

Authors: T Sawa; F Nishimura; H Ohyama; K Takahashi; S Takashiba; Y Murayama
Journal: Infect Immun Date: 1999-03 Impact factor: 3.441

2. Gingipains of Porphyromonas gingivalis modulate leukocyte adhesion molecule expression induced in human endothelial cells by ligation of CD99.

Authors: Peter L W Yun; Arthur A Decarlo; Neil Hunter
Journal: Infect Immun Date: 2006-03 Impact factor: 3.441

3. B and T lymphocytes are the primary sources of RANKL in the bone resorptive lesion of periodontal disease.

Authors: Toshihisa Kawai; Takashi Matsuyama; Yoshitaka Hosokawa; Seicho Makihira; Makoto Seki; Nadeem Y Karimbux; Reginaldo B Goncalves; Paloma Valverde; Serge Dibart; Yi-Ping Li; Leticia A Miranda; Cory W O Ernst; Yuichi Izumi; Martin A Taubman
Journal: Am J Pathol Date: 2006-09 Impact factor: 4.307

4. Immunohistological analysis of experimental gingivitis in humans.

Authors: G J Seymour; E Gemmell; L J Walsh; R N Powell
Journal: Clin Exp Immunol Date: 1988-01 Impact factor: 4.330

5. CD4(+) T cells and the proinflammatory cytokines gamma interferon and interleukin-6 contribute to alveolar bone loss in mice.

Authors: P J Baker; M Dixon; R T Evans; L Dufour; E Johnson; D C Roopenian
Journal: Infect Immun Date: 1999-06 Impact factor: 3.441

6. Diminished forkhead box P3/CD25 double-positive T regulatory cells are associated with the increased nuclear factor-kappaB ligand (RANKL+) T cells in bone resorption lesion of periodontal disease.

Authors: C W O Ernst; J E Lee; T Nakanishi; N Y Karimbux; T M B Rezende; P Stashenko; M Seki; M A Taubman; T Kawai
Journal: Clin Exp Immunol Date: 2007-03-09 Impact factor: 4.330

Phenotypic studies of cells from periodontal disease tissues.

1. In vitro induction of activation-induced cell death in lymphocytes from chronic periodontal lesions by exogenous Fas ligand.

2. Gingipains of Porphyromonas gingivalis modulate leukocyte adhesion molecule expression induced in human endothelial cells by ligation of CD99.

3. B and T lymphocytes are the primary sources of RANKL in the bone resorptive lesion of periodontal disease.

4. Immunohistological analysis of experimental gingivitis in humans.

5. CD4(+) T cells and the proinflammatory cytokines gamma interferon and interleukin-6 contribute to alveolar bone loss in mice.

6. Diminished forkhead box P3/CD25 double-positive T regulatory cells are associated with the increased nuclear factor-kappaB ligand (RANKL+) T cells in bone resorption lesion of periodontal disease.

7. Periodontal bone loss and immune characteristics of congenitally athymic and thymus cell-reconstituted athymic rats.

8. Phenotypic analyses of mononuclear cells recovered from healthy and diseased human periodontal tissues.

9. Selected Th1 and Th2 cytokine mRNA expression by CD4(+) T cells isolated from inflamed human gingival tissues.

10. Expression of SOFAT by T- and B-lineage cells may contribute to bone loss.