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Literature DB >> 6235990

Humoral and cellular immune response of the rat to immunization with bee venom.

Abstract

Lewis rats were immunized with bee venom allergen in Freund's complete adjuvant (FCA) or with FCA only. Animals immunized with bee venom developed specific IgG antibodies but no specific IgE antibodies were detected. Lymphocytes from lymph nodes when cultured with antigen in vitro showed an increased stimulation index from day 17 onwards. A concomitant augmentation of T suppressor cells was observed; the T helper/T suppressor cell ratio declined from 4.5:1 before immunization to 1:1 from day 5 onwards.

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Year: 1984 PMID： 6235990 PMCID： PMC1536134

Source DB: PubMed Journal: Clin Exp Immunol ISSN： 0009-9104 Impact factor: 4.330

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References

20 in total

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Authors: O Strannegård; I L Strannegård
Journal: Immunol Rev Date: 1978 Impact factor: 12.988

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Authors: J Forster; R Urbanek
Journal: Klin Wochenschr Date: 1979-04-17

10. Hapten-specific IgE antibody responses in mice. VII. Conversion of IgE "non-responder" strains to IgE "responders" by elimination of suppressor T cell activity.

Authors: N Chiorazzi; D A Fox; D H Katz
Journal: J Immunol Date: 1977-01 Impact factor: 5.422

Humoral and cellular immune response of the rat to immunization with bee venom.

Review 1. T lymphocyte numbers and function in human IgE-mediated allergy.

2. Enzyme-linked immunosorbent assay of allergen-specific IgG antibodies in bee sting allergic patients hyposensitized with pure bee venom.

3. Insect allergy: the state of the art.

Review 4. The significance of immunoglobulin E in reaginic hypersensitivity.

5. A new and simple radioimmunoassay method for the determination of IgE.

6. A double-blind study of the effectiveness and specificity of injecton therapy in ragweed hay fever.

Review 7. Cellular events in the IgE antibody response.

8. Enumeration of T cell subsets in atopic dermatitis using monoclonal antibodies.

Review 9. Mechanisms of reaginic hypersensitivity and immunotherapy.

10. Hapten-specific IgE antibody responses in mice. VII. Conversion of IgE "non-responder" strains to IgE "responders" by elimination of suppressor T cell activity.