Literature DB >> 6197262

Pathological vascularization of the coronary intima.

W J Cliff, G I Schoefl.   

Abstract

Secondary vascularization was found in the walls of diseased coronary arteries from 36 individuals who died after sudden cardiac arrest. In the series presented here, coronary arteries of hospital patients dying from all causes also showed a 100% incidence of pathological neovascularization. A process linking neovascularization and chronic inflammation to an ultimate infectious aetiology is suggested.

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Year:  1983        PMID: 6197262     DOI: 10.1002/9780470720813.ch12

Source DB:  PubMed          Journal:  Ciba Found Symp        ISSN: 0300-5208


  4 in total

1.  Endogenous regulation of angiogenesis in the rat aorta model. Role of vascular endothelial growth factor.

Authors:  R F Nicosia; Y J Lin; D Hazelton; X Qian
Journal:  Am J Pathol       Date:  1997-11       Impact factor: 4.307

2.  Immunohistochemical study of intimal microvessels in coronary atherosclerosis.

Authors:  Y Zhang; W J Cliff; G I Schoefl; G Higgins
Journal:  Am J Pathol       Date:  1993-07       Impact factor: 4.307

3.  Plasma protein insudation as an index of early coronary atherogenesis.

Authors:  Y Zhang; W J Cliff; G I Schoefl; G Higgins
Journal:  Am J Pathol       Date:  1993-08       Impact factor: 4.307

4.  Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipoproteins invade coronary intima via neovascularization from adventitial vasa vasorum, but not from the arterial lumen: a hypothesis.

Authors:  Vladimir M Subbotin
Journal:  Theor Biol Med Model       Date:  2012-04-10       Impact factor: 2.432

  4 in total

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