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15 in total

1. Inhibition of killer-target cell interaction by monoclonal anti-H-2 antibodies.

Authors: K F Lindahl; H Lemke
Journal: Eur J Immunol Date: 1979-07 Impact factor: 5.532

2. H-2 expression by lymphoid cells of different mouse strains: quantitative interaction of H-2 with monoclonal antibodies and their Fab fragments.

Authors: C J Hackett; B A Askonas
Journal: Immunology Date: 1981-02 Impact factor: 7.397

3. Target-cell recognition by cloned lines of influenza virus-specific cytotoxic T lymphocytes. Selective inhibition by a monoclonal H-2-specific antibody.

Authors: T J Braciale; C F Mojcik; V Hauptfeld
Journal: Immunogenetics Date: 1982-01 Impact factor: 2.846

4. Recognition of Db and Kb gene products by influenza-specific cytotoxic T cells.

Authors: A R Townsend; P M Taylor; A L Mellor; B A Askonas
Journal: Immunogenetics Date: 1983 Impact factor: 2.846

5. Product of a transferred H-2Ld gene acts as restriction element for LCMV-specific killer T cells.

Authors: A Orn; R S Goodenow; L Hood; P R Brayton; J G Woodward; R C Harmon; J A Frelinger
Journal: Nature Date: 1982-06-03 Impact factor: 49.962

6. Genetic and regulatory contributions of the major histocompatibility complex to the developing cytolytic T lymphocyte repertoire.

Authors: L A Sherman
Journal: J Immunol Date: 1982-04 Impact factor: 5.422

7. Expression of murine H-2Kb histocompatibility antigen in cells transformed with cloned H-2 genes.

Authors: A L Mellor; L Golden; E Weiss; H Bullman; J Hurst; E Simpson; R F James; A R Townsend; P M Taylor; W Schmidt; J Ferluga; L Leben; M Santamaria; G Atfield; H Festenstein; R A Flavell
Journal: Nature Date: 1982-08-05 Impact factor: 49.962

8. Monoclonal antibodies to hemagglutinin and to H-2 inhibit the cross-reactive cytotoxic T cell populations induced by influenza.

Authors: B A Askonas; R G Webster
Journal: Eur J Immunol Date: 1980-02 Impact factor: 5.532

3. Class I H-2d-restricted cytotoxic T lymphocytes recognize the neuraminidase glycoprotein of influenza virus subtype N1.

Authors: M Wysocka; C J Hackett
Journal: J Virol Date: 1990-03 Impact factor: 5.103

3 in total

Cytotoxic T-cell recognition of influenza-infected target cells varies in different H-2k mouse strains.

1. Inhibition of killer-target cell interaction by monoclonal anti-H-2 antibodies.

2. H-2 expression by lymphoid cells of different mouse strains: quantitative interaction of H-2 with monoclonal antibodies and their Fab fragments.

3. Target-cell recognition by cloned lines of influenza virus-specific cytotoxic T lymphocytes. Selective inhibition by a monoclonal H-2-specific antibody.

4. Recognition of Db and Kb gene products by influenza-specific cytotoxic T cells.

5. Product of a transferred H-2Ld gene acts as restriction element for LCMV-specific killer T cells.

6. Genetic and regulatory contributions of the major histocompatibility complex to the developing cytolytic T lymphocyte repertoire.

7. Expression of murine H-2Kb histocompatibility antigen in cells transformed with cloned H-2 genes.

8. Monoclonal antibodies to hemagglutinin and to H-2 inhibit the cross-reactive cytotoxic T cell populations induced by influenza.

9. Hybridoma cell lines secreting monoclonal antibodies to mouse H-2 and Ia antigens.

10. Influence of the major histocompatibility complex on the repertoire of allospecific cytolytic T lymphocytes.

Review 1. Recognition of influenza virus proteins by cytotoxic T lymphocytes.

2. Dk-restricted antiinfluenza cytotoxic T-cell clone loses one of its two alloreactivities.

3. Class I H-2d-restricted cytotoxic T lymphocytes recognize the neuraminidase glycoprotein of influenza virus subtype N1.