Literature DB >> 6102278

Autosomal dominant transmission and absence of HLA linkage in dexamethasone suppressible hyperaldosteronism.

M I New, S E Oberfield, L S Levine, B Dupont, M Pollack, J R Gill, F C Bartter.   

Abstract

Entities:  

Mesh:

Substances:

Year:  1980        PMID: 6102278     DOI: 10.1016/s0140-6736(80)92814-7

Source DB:  PubMed          Journal:  Lancet        ISSN: 0140-6736            Impact factor:   79.321


× No keyword cloud information.
  5 in total

1.  Variation of phenotype in patients with glucocorticoid remediable aldosteronism.

Authors:  L J Gates; A A MacConnachie; R P Lifton; N E Haites; N Benjamin
Journal:  J Med Genet       Date:  1996-01       Impact factor: 6.318

Review 2.  Low-renin hypertension of childhood.

Authors:  J DiMartino-Nardi; M I New
Journal:  Pediatr Nephrol       Date:  1987-01       Impact factor: 3.714

3.  Dexamethasone-suppressible hyperaldosteronism: pathophysiology, clinical aspects, and new insights into the pathogenesis.

Authors:  F Fallo; N Sonino; M Boscaro; D Armanini; F Mantero; H G Dörr; D Knorr; U Kuhnle
Journal:  Klin Wochenschr       Date:  1987-05-15

Review 4.  DIAGNOSIS OF ENDOCRINE DISEASE: 18-Oxocortisol and 18-hydroxycortisol: is there clinical utility of these steroids?

Authors:  Jacques W M Lenders; Tracy Ann Williams; Martin Reincke; Celso E Gomez-Sanchez
Journal:  Eur J Endocrinol       Date:  2017-09-13       Impact factor: 6.664

5.  Glucocorticoid-suppressible hyperaldosteronism results from hybrid genes created by unequal crossovers between CYP11B1 and CYP11B2.

Authors:  L Pascoe; K M Curnow; L Slutsker; J M Connell; P W Speiser; M I New; P C White
Journal:  Proc Natl Acad Sci U S A       Date:  1992-09-01       Impact factor: 11.205

  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.