Literature DB >> 6086854

Synchronization without active chemical synapses during hippocampal afterdischarges.

C P Taylor, F E Dudek.   

Abstract

Afterdischarges elicited by antidromic stimulation were studied in the CA1 region of transverse slices of rat hippocampus. The slices were incubated in a static bathing medium containing Mn2+ with lowered Ca2+ concentration, which blocked chemical synaptic transmission. Extracellular voltage recordings from micropipettes revealed afterdischarges of population spikes, which represent synchronized action potentials. Afterdischarge duration became longer with incubation time, occasionally lasting up to 9 s. Simultaneous recordings from three extracellular micropipettes indicated propagation of individual population spikes along the CA1 cell body layer during an afterdischarge. Intracellular recordings from pyramidal cells showed that action potentials were synchronized with population spikes. When initial action potentials of an afterdischarge were blocked by hyperpolarizing the impaled cell, subsequent action potentials remained synchronized with population spikes, indicating a synchronizing interaction between neurons. Intrasomatic neuronal recordings sometimes revealed partial action potentials that were also synchronized with population spikes. During afterdischarges, relatively weak (10 mV) hyperpolarization blocked both full and partial action potentials. These observations suggest that synchronized action potentials and partial spikes were initiated near the soma. Intracellular voltage recordings were referenced to a local extracellular microelectrode in order to determine more accurately the transmembrane potential in the presence of large population spikes .54). Such neuronal recordings revealed brief transmembrane depolarizations that occurred synchronously with population spikes. Large injected hyperpolarizing currents did not affect the brief transmembrane depolarizations. Similar differential voltage recordings across glial cell membranes did not show measurable brief transmembrane depolarizations during population spikes. However, slow glial depolarizations during afterdischarges indicated prolonged K+ accumulation and clearance in the extracellular space, which probably contributed to the hyperexcitability of pyramidal cells. During an afterdischarge, extracellular potentials were recorded simultaneously from six micropipettes arranged perpendicularly to the stratum pyramidale. Current source-density analysis indicated a sink in the cell body layer during population spikes and a corresponding source in distal dendrites. It is concluded that an electrical field effect depolarizes and thus synchronizes neurons during these afterdischarges.(ABSTRACT TRUNCATED AT 400 WORDS)

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Year:  1984        PMID: 6086854     DOI: 10.1152/jn.1984.52.1.143

Source DB:  PubMed          Journal:  J Neurophysiol        ISSN: 0022-3077            Impact factor:   2.714


  18 in total

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2.  Cortical hyperpolarization-activated depolarizing current takes part in the generation of focal paroxysmal activities.

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Journal:  Proc Natl Acad Sci U S A       Date:  2002-06-27       Impact factor: 11.205

3.  Focal generation of paroxysmal fast runs during electrographic seizures.

Authors:  Sofiane Boucetta; Sylvain Chauvette; Maxim Bazhenov; Igor Timofeev
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4.  Optical recording of epileptiform voltage changes in the neocortical slice.

Authors:  B Albowitz; U Kuhnt; L Ehrenreich
Journal:  Exp Brain Res       Date:  1990       Impact factor: 1.972

5.  Persistent sodium current drives conditional pacemaking in CA1 pyramidal neurons under muscarinic stimulation.

Authors:  Jason Yamada-Hanff; Bruce P Bean
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6.  Ictal epileptiform activity is facilitated by hippocampal GABAA receptor-mediated oscillations.

Authors:  R Köhling; M Vreugdenhil; E Bracci; J G Jefferys
Journal:  J Neurosci       Date:  2000-09-15       Impact factor: 6.167

Review 7.  Calcium and epileptogenesis.

Authors:  U Heinemann; B Hamon
Journal:  Exp Brain Res       Date:  1986       Impact factor: 1.972

8.  Physiological and morphological characterization of dentate granule cells in the p35 knock-out mouse hippocampus: evidence for an epileptic circuit.

Authors:  Leena S Patel; H Jürgen Wenzel; Philip A Schwartzkroin
Journal:  J Neurosci       Date:  2004-10-13       Impact factor: 6.167

9.  Propagation velocity of epileptiform activity in the hippocampus.

Authors:  J Holsheimer; F H Lopes da Silva
Journal:  Exp Brain Res       Date:  1989       Impact factor: 1.972

10.  The effect of calcium removal on the suppression by adenosine of epileptiform activity in the hippocampus: demonstration of desensitization.

Authors:  H Hosseinzadeh; T W Stone
Journal:  Br J Pharmacol       Date:  1994-05       Impact factor: 8.739

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