Literature DB >> 5935042

The cellular basis for the defect in haemopoiesis in flexed-tailed mice. I. The nature and persistance of the defect.

M W Thompson, E A McCulloch, L Siminovitch, J E Till.   

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Year:  1966        PMID： 5935042     DOI： 10.1111/j.1365-2141.1966.tb05619.x

Source DB:  PubMed          Journal:  Br J Haematol        ISSN： 0007-1048            Impact factor:   6.998


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  4 in total

1.  A mutation in a mitochondrial transmembrane protein is responsible for the pleiotropic hematological and skeletal phenotype of flexed-tail (f/f) mice.

Authors:  M D Fleming; D R Campagna; J N Haslett; C C Trenor; N C Andrews
Journal:  Genes Dev       Date:  2001-03-15       Impact factor: 11.361

2.  BMP4/Smad5 dependent stress erythropoiesis is required for the expansion of erythroid progenitors during fetal development.

Authors:  Prashanth Porayette; Robert F Paulson
Journal:  Dev Biol       Date:  2008-02-15       Impact factor: 3.582

Review 3.  Application of marrow grafts in human disease.

Authors:  G W Santos
Journal:  Am J Pathol       Date:  1971-12       Impact factor: 4.307

4.  Negative autoregulation by FAS mediates robust fetal erythropoiesis.

Authors:  Merav Socolovsky; Michael Murrell; Ying Liu; Ramona Pop; Ermelinda Porpiglia; Andre Levchenko
Journal:  PLoS Biol       Date:  2007-10       Impact factor: 8.029
  4 in total

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