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103 in total

1. Ultrastructural study of Betz cells in the primary motor cortex of the human brain.

Authors: S Sasaki; M Iwata
Journal: J Anat Date: 2001-12 Impact factor: 2.610

2. Postsynaptic degeneration as revealed by PSD-95 reduction occurs after advanced Aβ and tau pathology in transgenic mouse models of Alzheimer's disease.

Authors: Charles Y Shao; Suzanne S Mirra; Hameetha B R Sait; Todd C Sacktor; Einar M Sigurdsson
Journal: Acta Neuropathol Date: 2011-06-01 Impact factor: 17.088

3. Ultrastructure of the neuropil threads in the Alzheimer brain: their dendritic origin and accumulation in the senile plaques.

Authors: H Yamaguchi; Y Nakazato; M Shoji; Y Ihara; S Hirai
Journal: Acta Neuropathol Date: 1990 Impact factor: 17.088

4. Formation of Hirano bodies induced by expression of an actin cross-linking protein with a gain-of-function mutation.

Authors: Andrew Maselli; Ruth Furukawa; Susanne A M Thomson; Richard C Davis; Marcus Fechheimer
Journal: Eukaryot Cell Date: 2003-08

5. Elucidation of three-dimensional ultrastructure of Hirano bodies by the quick-freeze, deep-etch and replica method.

Authors: N Izumiyama; K Ohtsubo; T Tachikawa; H Nakamura
Journal: Acta Neuropathol Date: 1991 Impact factor: 17.088

6. Ultrastructural identification of neurofibrillary tangles in the spinal cords in Guamanian amyotrophic lateral sclerosis and parkinsonism-dementia complex on Guam.

Authors: S Kato; A Hirano; J F Llena; H Ito; S H Yen
Journal: Acta Neuropathol Date: 1992 Impact factor: 17.088

The fine structure of some intraganglionic alterations. Neurofibrillary tangles, granulovacuolar bodies and "rod-like" structures as seen in Guam amyotrophic lateral sclerosis and parkinsonism-dementia complex.

1. Ultrastructural study of Betz cells in the primary motor cortex of the human brain.

2. Postsynaptic degeneration as revealed by PSD-95 reduction occurs after advanced Aβ and tau pathology in transgenic mouse models of Alzheimer's disease.

3. Ultrastructure of the neuropil threads in the Alzheimer brain: their dendritic origin and accumulation in the senile plaques.

4. Formation of Hirano bodies induced by expression of an actin cross-linking protein with a gain-of-function mutation.

5. Elucidation of three-dimensional ultrastructure of Hirano bodies by the quick-freeze, deep-etch and replica method.

6. Ultrastructural identification of neurofibrillary tangles in the spinal cords in Guamanian amyotrophic lateral sclerosis and parkinsonism-dementia complex on Guam.

7. Straight and paired helical filaments in Alzheimer disease have a common structural unit.

8. Rod-shaped filamentous inclusions and other ultrastructural features in a cerebellar astrocytoma.

9. Spongiform alterations in brain biopsies of presenile dementia.

10. Reappraisal of the fine structure of Alzheimer's neurofibrillary tangles.