Literature DB >> 5479631

Ribonuclease-inhibitor system abnormality in dystrophic mouse skeletal muscle.

B W Little, W L Meyer.   

Abstract

Skeletal muscle extracts from mice with muscular dystrophy contain severalfold higher than normal levels of free alkaline ribonuclease II activity and none of the free ribonuclease inhibitor normally present. This abnormal pattern is not seen in heart or liver extracts from dystrophic mice.

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Year:  1970        PMID： 5479631     DOI： 10.1126/science.170.3959.747

Source DB:  PubMed          Journal:  Science        ISSN： 0036-8075            Impact factor:   47.728


  4 in total

1.  The expression of glycogen phosphorylase in normal and dystrophic muscle.

Authors:  D M Leyland; R J Beynon
Journal:  Biochem J       Date:  1991-08-15       Impact factor: 3.857

2.  Secretion of ribonucleases by normal and immortalized cells grown in serum-free culture conditions.

Authors:  M Moenner; E Hatzi; J Badet
Journal:  In Vitro Cell Dev Biol Anim       Date:  1997 Jul-Aug       Impact factor: 2.416

3.  The ribonucleases of bovine skeletal muscle.

Authors:  G E Davies; T P Karpetsky; C C Levy
Journal:  Biochem J       Date:  1980-08-01       Impact factor: 3.857

4.  The primary structure of human ribonuclease/angiogenin inhibitor (RAI) discloses a novel highly diversified protein superfamily with a common repetitive module.

Authors:  R Schneider; E Schneider-Scherzer; M Thurnher; B Auer; M Schweiger
Journal:  EMBO J       Date:  1988-12-20       Impact factor: 11.598
  4 in total

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