Literature DB >> 502347

[Current problems of glomerulonephritis (author's transl)].

W Thoenes.   

Abstract

Setting out with the theory of glomerulonephritis from Volhard and Fahr (1914) and the fundamental patho-anatomical examinations on this subject by Theodor Fahr (1925, 1934) the actual problems of glomerulonephritis are described. Progress in nephropathology and immunopathology which was partly conditioned by progress in the methodical fields has come to the result that, nowadays, an appropriate characterization of glomerulonephritic diseases is only possible by the synopsis of the three categories: Immunopathogenesis, pathomorphology and clinic. Each of these categories has its own classification. The nosology of the various GN types results from the (constant or variable) combination of the components of these three systems. By this means, some known types of glomerulonephritis were confirmed, some were newly defined, and new entities with variable clinical relevance were layed bare. Because immunopathogenetic events very often induce characteristic structural alterations in the glomeruli, it is the pathomorphological nomenclature which -- as pars pro toto -- still is the best suited for common interchange.

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Year:  1979        PMID: 502347     DOI: 10.1007/bf01477985

Source DB:  PubMed          Journal:  Klin Wochenschr        ISSN: 0023-2173


  45 in total

1.  Dense deposit disease: a variant of membranoproliferative glomerulonephritis.

Authors:  R Habib; M C Gubler; C Loirat; H B Mäiz; M Levy
Journal:  Kidney Int       Date:  1975-04       Impact factor: 10.612

2.  [Membrane-proliferative glomerulonephritis. Immunologic and pathogenetic aspects].

Authors:  H P Seelig; R Seelig
Journal:  Dtsch Med Wochenschr       Date:  1977-06-24       Impact factor: 0.628

3.  Correlations between morphologic and clinical features in idiopathic perimembranous glomerulonephritis. A study on 403 renal biopsies of 367 patients.

Authors:  H V Gärtner; T Watanabe; V Ott; A Adam; A Bohle; H H Edel; R Kluthe; E Renner; F Scheler; R M Schmülling; H G Sieberth
Journal:  Curr Top Pathol       Date:  1977

4.  [Immunohistological observations in minimal changes-, and focal sclerosing glomerulopathy with nephrotic syndrome (author's transl)].

Authors:  G H Thoenes
Journal:  Klin Wochenschr       Date:  1974-04-15

5.  Idiopathic recurrent macroscopic hematuria and mesangial IgA-IgG deposits in children (Berger's disease).

Authors:  M Levy; H Beaufils; M C Gubler; R Habib
Journal:  Clin Nephrol       Date:  1972 Mar-Apr       Impact factor: 0.975

6.  IgA glomerular deposits in renal disease.

Authors:  J Berger
Journal:  Transplant Proc       Date:  1969-12       Impact factor: 1.066

7.  [Focal glomerular lesions in idiopathic nephrotic syndrome of childhood. Observations of 49 cases].

Authors:  R Habib; M C Gubler
Journal:  Nephron       Date:  1971       Impact factor: 2.847

8.  Pathogenesis of lipoid nephrosis: a disorder of T-cell function.

Authors:  R J Shalhoub
Journal:  Lancet       Date:  1974-09-07       Impact factor: 79.321

9.  Inhibition of lymphocyte blastogenesis by plasma of patients with minimal-change nephrotic syndrome.

Authors:  A V Moorthy; S W Zimmerman; P M Burkholder
Journal:  Lancet       Date:  1976-05-29       Impact factor: 79.321

10.  In vitro demonstration of a particular affinity of glomerular basement membrane and collagen for DNA. A possible basis for a local formation of DNA-anti-DNA complexes in systemic lupus erythematosus.

Authors:  S Izui; P H Lambert; P A Miescher
Journal:  J Exp Med       Date:  1976-08-01       Impact factor: 14.307

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  1 in total

1.  The mesangium and glomerulonephritis.

Authors:  R B Sterzel; D H Lovett; H D Stein; M Kashgarian
Journal:  Klin Wochenschr       Date:  1982-09-15
  1 in total

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