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123 in total

1. Free heme and the polymerization of sickle cell hemoglobin.

Authors: Veselina V Uzunova; Weichun Pan; Oleg Galkin; Peter G Vekilov
Journal: Biophys J Date: 2010-09-22 Impact factor: 4.033

2. Auto-oxidation of hemoglobin in plasma.

Authors: M Furlan; H Bachofen
Journal: Experientia Date: 1976-01-15

3. Astrocyte-specific heme oxygenase-1 hyperexpression attenuates heme-mediated oxidative injury.

Authors: Luna Benvenisti-Zarom; Raymond F Regan
Journal: Neurobiol Dis Date: 2007-03-24 Impact factor: 5.996

4. Targeting heme oxygenase after intracerebral hemorrhage.

Authors: Jing Chen-Roetling; Xiangping Lu; Raymond F Regan
Journal: Ther Targets Neurol Dis Date: 2015-01-03

5. Systemic hemin therapy attenuates blood-brain barrier disruption after intracerebral hemorrhage.

Authors: Xiangping Lu; Jing Chen-Roetling; Raymond F Regan
Journal: Neurobiol Dis Date: 2014-06-18 Impact factor: 5.996

6. Identification of Mechanism-Based Inactivation in P450-Catalyzed Cyclopropanation Facilitates Engineering of Improved Enzymes.

Authors: Hans Renata; Russell D Lewis; Michael J Sweredoski; Annie Moradian; Sonja Hess; Z Jane Wang; Frances H Arnold
Journal: J Am Chem Soc Date: 2016-09-14 Impact factor: 15.419

Exchange of heme among hemoglobins and between hemoglobin and albumin.

1. Free heme and the polymerization of sickle cell hemoglobin.

2. Auto-oxidation of hemoglobin in plasma.

3. Astrocyte-specific heme oxygenase-1 hyperexpression attenuates heme-mediated oxidative injury.

4. Targeting heme oxygenase after intracerebral hemorrhage.

5. Systemic hemin therapy attenuates blood-brain barrier disruption after intracerebral hemorrhage.

6. Identification of Mechanism-Based Inactivation in P450-Catalyzed Cyclopropanation Facilitates Engineering of Improved Enzymes.

7. Heme-protein fission under nondenaturing conditions.

8. Increasing expression of H- or L-ferritin protects cortical astrocytes from hemin toxicity.

9. Disposal of plasma heme in normal man and patients with intravascular hemolysis.

10. Haem transport to the liver by haemopexin. Receptor-mediated uptake with recycling of the protein.