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Literature DB >> 4711805

Axon outgrowth enhanced by a previous nerve injury.

I G McQuarrie, B Grafstein.

Abstract

Entities: Disease

Mesh：

Year: 1973 PMID： 4711805 DOI： 10.1001/archneur.1973.00490250071008

Source DB: PubMed Journal: Arch Neurol ISSN： 0003-9942

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62 in total

1. Nature of the retrograde signal from injured nerves that induces interleukin-6 mRNA in neurons.

Authors: P G Murphy; L S Borthwick; R S Johnston; G Kuchel; P M Richardson
Journal: J Neurosci Date: 1999-05-15 Impact factor: 6.167

2. Interleukin-17A increases neurite outgrowth from adult postganglionic sympathetic neurons.

Authors: Susan P Chisholm; Andrea L Cervi; Simrin Nagpal; Alan E Lomax
Journal: J Neurosci Date: 2012-01-25 Impact factor: 6.167

3. Chronic enhancement of the intrinsic growth capacity of sensory neurons combined with the degradation of inhibitory proteoglycans allows functional regeneration of sensory axons through the dorsal root entry zone in the mammalian spinal cord.

Authors: Michael P Steinmetz; Kevin P Horn; Veronica J Tom; Jared H Miller; Sarah A Busch; Dileep Nair; Daniel J Silver; Jerry Silver
Journal: J Neurosci Date: 2005-08-31 Impact factor: 6.167

4. The unusual response of serotonergic neurons after CNS Injury: lack of axonal dieback and enhanced sprouting within the inhibitory environment of the glial scar.

Authors: Alicia L Hawthorne; Hongmei Hu; Bornali Kundu; Michael P Steinmetz; Christi J Wylie; Evan S Deneris; Jerry Silver
Journal: J Neurosci Date: 2011-04-13 Impact factor: 6.167

Axon outgrowth enhanced by a previous nerve injury.

1. Nature of the retrograde signal from injured nerves that induces interleukin-6 mRNA in neurons.

2. Interleukin-17A increases neurite outgrowth from adult postganglionic sympathetic neurons.

3. Chronic enhancement of the intrinsic growth capacity of sensory neurons combined with the degradation of inhibitory proteoglycans allows functional regeneration of sensory axons through the dorsal root entry zone in the mammalian spinal cord.

4. The unusual response of serotonergic neurons after CNS Injury: lack of axonal dieback and enhanced sprouting within the inhibitory environment of the glial scar.

5. STAT3 integrates cytokine and neurotrophin signals to promote sympathetic axon regeneration.

6. Injury-induced HDAC5 nuclear export is essential for axon regeneration.

7. Depolarization and electrical stimulation enhance in vitro and in vivo sensory axon growth after spinal cord injury.

Review 8. Intra-axonal mechanisms driving axon regeneration.

9. CNS regeneration: only on one condition.

10. A conditioning lesion provides selective protection in a rat model of Amyotrophic Lateral Sclerosis.