Literature DB >> 470726

Indomethacin-induced hyperkalaemia.

E P MacCarthy, G W Frost, G S Strokes.   

Abstract

A patient with mild chronic renal failure developed significant hyperkalaemia while receiving indomethacin therapy. The hyperkalaemia, which was reversed by discontinuing indomethacin, is attributed to a drug-induced defect in the cellular uptake of potassium.

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Year:  1979        PMID: 470726     DOI: 10.5694/j.1326-5377.1979.tb127061.x

Source DB:  PubMed          Journal:  Med J Aust        ISSN: 0025-729X            Impact factor:   7.738


  3 in total

1.  Salt substitutes and potassium intake.

Authors:  J D Swales
Journal:  BMJ       Date:  1991-11-02

2.  Increase in plasma potassium concentration following indomethacin administration: absence of a role for membrane Na-K ATPase.

Authors:  A Padhani; K Turaihi; D Junglee; R K Menon; P Dandona
Journal:  Agents Actions       Date:  1987-10

Review 3.  Nonsteroidal anti-inflammatory drugs and acute renal failure in the elderly. A risk-benefit assessment.

Authors:  W Ailabouni; G Eknoyan
Journal:  Drugs Aging       Date:  1996-11       Impact factor: 3.923

  3 in total

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