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Literature DB >> 465354

Mechanism of desferrioxamine-induced iron excretion in thalassaemia.

C Hershko, E A Rachmilewitz.

Abstract

Entities: Chemical Disease

Mesh：

Substances：

Year: 1979 PMID： 465354 DOI： 10.1111/j.1365-2141.1979.tb03704.x

Source DB: PubMed Journal: Br J Haematol ISSN： 0007-1048 Impact factor: 6.998

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12 in total

1. Timed non-transferrin bound iron determinations probe the origin of chelatable iron pools during deferiprone regimens and predict chelation response.

Authors: Yesim Aydinok; Patricia Evans; Chantal Y Manz; John B Porter
Journal: Haematologica Date: 2011-12-16 Impact factor: 9.941

2. Future alternative therapies for β-thalassemia.

Authors: Stefano Rivella; Eliezer Rachmilewitz
Journal: Expert Rev Hematol Date: 2009-12-01 Impact factor: 2.929

3. Hepcidin as a therapeutic tool to limit iron overload and improve anemia in β-thalassemic mice.

Authors: Sara Gardenghi; Pedro Ramos; Maria Franca Marongiu; Luca Melchiori; Laura Breda; Ella Guy; Kristen Muirhead; Niva Rao; Cindy N Roy; Nancy C Andrews; Elizabeta Nemeth; Antonia Follenzi; Xiuli An; Narla Mohandas; Yelena Ginzburg; Eliezer A Rachmilewitz; Patricia J Giardina; Robert W Grady; Stefano Rivella
Journal: J Clin Invest Date: 2010-11-22 Impact factor: 14.808

Mechanism of desferrioxamine-induced iron excretion in thalassaemia.

1. Timed non-transferrin bound iron determinations probe the origin of chelatable iron pools during deferiprone regimens and predict chelation response.

2. Future alternative therapies for β-thalassemia.

3. Hepcidin as a therapeutic tool to limit iron overload and improve anemia in β-thalassemic mice.

4. Iron overload: pathogenesis and treatment with chelating agents.

Review 5. Regulation of iron absorption in hemoglobinopathies.

6. A mathematical model of iron metabolism.

Review 7. Ineffective erythropoiesis and thalassemias.

8. Intravenous and subcutaneous desferrioxamine therapy in children with severe iron overload.

9. Iron metabolism in thalassemia and sickle cell disease.

Review 10. Iron overload in patients with myelodysplastic syndromes.