Literature DB >> 4637300

Skin lesions produced by Staphylococcus aureus exfoliatin in hairless mice.

F A Kapral, M M Miller.   

Abstract

Exfoliatin caused skin loss in hairless mutant mice only while they lacked hair, but older mice were more resistant to this effect than neonates.

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Year:  1972        PMID: 4637300      PMCID: PMC422623          DOI: 10.1128/iai.6.5.877-879.1972

Source DB:  PubMed          Journal:  Infect Immun        ISSN: 0019-9567            Impact factor:   3.441


  3 in total

1.  The staphylococcal scalded-skin syndrome: isolation and partial characterization of the exfoliative toxin.

Authors:  M E Melish; L A Glasgow; M D Turner
Journal:  J Infect Dis       Date:  1972-02       Impact factor: 5.226

2.  Product of Staphylococcus aureus responsible for the scalded-skin syndrome.

Authors:  F A Kapral; M M Miller
Journal:  Infect Immun       Date:  1971-11       Impact factor: 3.441

3.  Toxic epidermal necrolysis produced by an extracellular product of Staphylococcus aureus.

Authors:  J P Arbuthnott; J Kent; A Lyell; C G Gemmell
Journal:  Br J Dermatol       Date:  1971-08       Impact factor: 9.302

  3 in total
  4 in total

1.  Staphylococcal scalded skin syndrome: potentiation by immunosuppression in mice; toxin-mediated exfoliation in a healthy adult.

Authors:  B B Wiley; S Allman; M Rogolsky; C W Norden; L A Glasgow
Journal:  Infect Immun       Date:  1974-04       Impact factor: 3.441

2.  Genetic control of immune response to staphylococcal exfoliative toxin A in mice.

Authors:  K Machida; S Sakurai; I Kondo; S Ikawa
Journal:  Infect Immun       Date:  1987-11       Impact factor: 3.441

Review 3.  Clinical, microbial, and biochemical aspects of the exfoliative toxins causing staphylococcal scalded-skin syndrome.

Authors:  S Ladhani; C L Joannou; D P Lochrie; R W Evans; S M Poston
Journal:  Clin Microbiol Rev       Date:  1999-04       Impact factor: 26.132

4.  New type of exfoliatin obtained from staphylococcal strains, belonging to phage groups other than group II, isolated from patients with impetigo and Ritter's disease.

Authors:  I Kondo; S Sakurai; Y Sarai
Journal:  Infect Immun       Date:  1974-10       Impact factor: 3.441

  4 in total

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