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Literature DB >> 4637153

Hypercalcaemia, hypophosphataemia, and inability to excrete hydrogen ions.

H Arcila, J Chávez de los Ríos, R Van Dyck, E Zorrilla.

Abstract

Investigation of a patient with hypercalcaemia, hypophosphataemia, and nephrocalcinosis failed to lead to a clear diagnosis. Neither primary hyperparathyroidism nor primary incomplete renal tubular acidosis could explain all the biochemical features, and it seems that more than one fundamental abnormality may have been present.

Entities: Chemical Disease Species

Mesh：

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Year: 1972 PMID： 4637153 PMCID： PMC1786635 DOI： 10.1136/bmj.4.5837.400

Source DB: PubMed Journal: Br Med J ISSN： 0007-1447

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References

8 in total

1. TUBULAR REABSORPTION OF PHOSPHATE IN PRIMARY HYPERPARATHYROIDISM--BEFORE AND AFTER ADMINISTRATION OF PARATHYROID HORMONE.

Authors: K L BECKER; D C PURNELL; J D JONES
Journal: J Clin Endocrinol Metab Date: 1964-04 Impact factor: 5.958

2. RAPID CALCIUM INFUSION TEST FOR HYPERPARATHYROIDISM. FURTHER EXPERIENCES.

Authors: R S GOLDSMITH; M FORLAND
Journal: Arch Intern Med Date: 1964-04

3. Fanconi syndrome (adult type) developing secondary to the ingestion of outdated tetracycline.

Authors: J M GROSS
Journal: Ann Intern Med Date: 1963-03 Impact factor: 25.391

4. Renal tubular acidosis and renal potassium wasting acquired as a result of hypercalcemic nephropathy.

Authors: T FERRIS; M KASHGARIAN; H LEVITIN; I BRANDT; F H EPSTEIN
Journal: N Engl J Med Date: 1961-11-09 Impact factor: 91.245

5. The renal excretion of hydrogen ion in renal tubular acidosis. I. quantitative assessment of the response to ammonium chloride as an acid load.

Authors: J R ELKINTON; E J HUTH; G D WEBSTER; R A McCANCE
Journal: Am J Med Date: 1960-10 Impact factor: 4.965

Hypercalcaemia, hypophosphataemia, and inability to excrete hydrogen ions.

1. TUBULAR REABSORPTION OF PHOSPHATE IN PRIMARY HYPERPARATHYROIDISM--BEFORE AND AFTER ADMINISTRATION OF PARATHYROID HORMONE.

2. RAPID CALCIUM INFUSION TEST FOR HYPERPARATHYROIDISM. FURTHER EXPERIENCES.

3. Fanconi syndrome (adult type) developing secondary to the ingestion of outdated tetracycline.

4. Renal tubular acidosis and renal potassium wasting acquired as a result of hypercalcemic nephropathy.

5. The renal excretion of hydrogen ion in renal tubular acidosis. I. quantitative assessment of the response to ammonium chloride as an acid load.

6. The excretion of acid in renal disease.

7. Defects of water reabsorption and of hydrogen-ion excretion by the renal tubules in hyperparathyroidism.

8. Evidence for a direct effect of parathyroid hormone on urinary acidification.