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Review 1. Redox-dependent impairment of vascular function in sickle cell disease.

Authors: Mutay Aslan; Bruce A Freeman
Journal: Free Radic Biol Med Date: 2007-08-31 Impact factor: 7.376

2. Isolation and partial characterization of antibody- and globin-enriched complexes from membranes of dense human erythrocytes.

Authors: R Kannan; J Yuan; P S Low
Journal: Biochem J Date: 1991-08-15 Impact factor: 3.857

3. Hemolysis-associated endothelial dysfunction mediated by accelerated NO inactivation by decompartmentalized oxyhemoglobin.

Authors: Peter C Minneci; Katherine J Deans; Huang Zhi; Peter S T Yuen; Robert A Star; Steven M Banks; Alan N Schechter; Charles Natanson; Mark T Gladwin; Steven B Solomon
Journal: J Clin Invest Date: 2005-11-17 Impact factor: 14.808

4. Lactate dehydrogenase as a biomarker of hemolysis-associated nitric oxide resistance, priapism, leg ulceration, pulmonary hypertension, and death in patients with sickle cell disease.

Authors: Gregory J Kato; Vicki McGowan; Roberto F Machado; Jane A Little; James Taylor; Claudia R Morris; James S Nichols; Xunde Wang; Mirjana Poljakovic; Sidney M Morris; Mark T Gladwin
Journal: Blood Date: 2005-11-15 Impact factor: 22.113

Review 5. Nutritional support in sickle cell anemia: theoretical considerations.

Authors: C O Enwonwu
Journal: J Natl Med Assoc Date: 1988-02 Impact factor: 1.798

6. Inheritance of the Bantu/Benin haplotype causes less severe hemolytic and oxidative stress in sickle cell anemia patients treated with hydroxycarbamide.

Authors: Jéssika V Okumura; Danilo G H Silva; Lidiane S Torres; Edis Belini-Junior; Willian M Barberino; Renan G Oliveira; Gisele C S Carrocini; Gabriela B Gelaleti; Clarisse L C Lobo; Claudia R Bonini-Domingos
Journal: J Hum Genet Date: 2016-03-10 Impact factor: 3.172

7. Excessive binding of natural anti-alpha-galactosyl immunoglobin G to sickle erythrocytes may contribute to extravascular cell destruction.

Authors: U Galili; M R Clark; S B Shohet
Journal: J Clin Invest Date: 1986-01 Impact factor: 14.808

Hemolysis in sickle cell disease.