Literature DB >> 3970322

Dysphagia and lower esophageal sphincter abnormalities after proximal gastric vagotomy.

M Guelrud, V Zambrano-Rincones, C Simon, G Gomez, A Salinas, A Toledano, J Rudick.   

Abstract

We studied 96 patients subjected to elective proximal gastric vagotomy for intractable duodenal ulceration. Dysphagia was a frequent finding and occurred in 32 percent. It appeared in the immediate postoperative period and usually lasted for 1 to 2 months without any abnormalities in lower esophageal sphincter function. In five patients, dysphagia was severe and, although transient, was associated with changes in lower esophageal function simulating those observed in achalasia. The mechanism of these motor abnormalities is probably due to a reversible neuromuscular dysfunction of the lower esophageal sphincter.

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Year:  1985        PMID: 3970322     DOI: 10.1016/s0002-9610(85)80076-3

Source DB:  PubMed          Journal:  Am J Surg        ISSN: 0002-9610            Impact factor:   2.565


  4 in total

1.  Development of achalasia secondary to laparoscopic Nissen fundoplication.

Authors:  Nicholas Stylopoulos; Cheryl J Bunker; David W Rattner
Journal:  J Gastrointest Surg       Date:  2002 May-Jun       Impact factor: 3.452

2.  Achalasia-like syndrome presenting after highly selective vagotomy.

Authors:  T J Duntemann; D M Dresner
Journal:  Dig Dis Sci       Date:  1995-09       Impact factor: 3.199

3.  Should it be parietal cell vagotomy or selective vagotomy-antrectomy for treatment of duodenal ulcer? A progress report.

Authors:  P H Jordan; J Thornby
Journal:  Ann Surg       Date:  1987-05       Impact factor: 12.969

4.  Indications for parietal cell vagotomy without drainage in gastrointestinal surgery.

Authors:  P H Jordan
Journal:  Ann Surg       Date:  1989-07       Impact factor: 12.969

  4 in total

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