Literature DB >> 3966007

Defective [U-14 C] palmitic acid oxidation in Duchenne muscular dystrophy.

J E Carroll, B J Norris, M H Brooke.   

Abstract

Compared with normal skeletal muscle, muscle from patients with Duchenne dystrophy had decreased [U-14 C] palmitic acid oxidation. [1-14 C] palmitic acid oxidation was normal. These results may indicate a defect in intramitochondrial fatty acid oxidation.

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Year:  1985        PMID： 3966007     DOI： 10.1212/wnl.35.1.96

Source DB:  PubMed          Journal:  Neurology        ISSN： 0028-3878            Impact factor:   9.910


  4 in total

Review 1.  Effect of exercise on serum enzyme activities in humans.

Authors:  T D Noakes
Journal:  Sports Med       Date:  1987 Jul-Aug       Impact factor: 11.136

2.  Selective PPARδ Modulators Improve Mitochondrial Function: Potential Treatment for Duchenne Muscular Dystrophy (DMD).

Authors:  Bharat Lagu; Arthur F Kluge; Effie Tozzo; Ross Fredenburg; Eric L Bell; Matthew M Goddeeris; Peter Dwyer; Andrew Basinski; Ramesh S Senaiar; Mahaboobi Jaleel; Nirbhay Kumar Tiwari; Sunil K Panigrahi; Narasimha Rao Krishnamurthy; Taisuke Takahashi; Michael A Patane
Journal:  ACS Med Chem Lett       Date:  2018-07-31       Impact factor: 4.345

3.  Adenylosuccinic acid therapy ameliorates murine Duchenne Muscular Dystrophy.

Authors:  Cara A Timpani; Craig A Goodman; Christos G Stathis; Jason D White; Kamel Mamchaoui; Gillian Butler-Browne; Nuri Gueven; Alan Hayes; Emma Rybalka
Journal:  Sci Rep       Date:  2020-01-24       Impact factor: 4.379

Review 4.  Metabogenic and Nutriceutical Approaches to Address Energy Dysregulation and Skeletal Muscle Wasting in Duchenne Muscular Dystrophy.

Authors:  Emma Rybalka; Cara A Timpani; Christos G Stathis; Alan Hayes; Matthew B Cooke
Journal:  Nutrients       Date:  2015-11-26       Impact factor: 5.717
  4 in total

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