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Literature DB >> 3944020

Sex steroid-induced changes in collagen of the prostate and seminal vesicle of rats.

N Srinivasan, M M Aruldhas, P Govindarajulu.

Abstract

The effects of androgen alone or in combination with estrogen or prolactin on the collagen of the prostate and seminal vesicles were studied in prepubertal and adult rats. Castration decreased the collagen content of the male accessory sex organs of both prepubertal and adult rats. Androgens showed stimulatory effects in castrated rats irrespective of the age. However, in intact animals, the stimulatory effects of androgens were evident only before puberty. Only the seminal vesicle of prepubertal rats responded to the stimulatory effect of estrogen given along with androgens. Prolactin did not elicit any appreciable effect either in the prostate or the seminal vesicles when administered along with androgens.

Entities: Chemical Gene Species

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Year: 1986 PMID： 3944020 DOI： 10.1002/j.1939-4640.1986.tb00869.x

Source DB: PubMed Journal: J Androl ISSN： 0196-3635

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Cited

6 in total

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Journal: Mol Imaging Radionucl Ther Date: 2011-12-01

6. Testosterone reduces knee passive range of motion and expression of relaxin receptor isoforms via 5α-dihydrotestosterone and androgen receptor binding.

Authors: Firouzeh Dehghan; Sekaran Muniandy; Ashril Yusof; Naguib Salleh
Journal: Int J Mol Sci Date: 2014-03-17 Impact factor: 5.923

6 in total

Sex steroid-induced changes in collagen of the prostate and seminal vesicle of rats.

1. Effect of exogenous testosterone, finasteride, and castration on serum level of thyroxin.

2. Altered prostate epithelial development in mice lacking the androgen receptor in stromal fibroblasts.

3. Assessing the antiandrogenic properties of propyl paraben using the Hershberger bioassay.

4. Sex, collagen expression, and anterior cruciate ligament strength in rats.

5. The Effect of Patient Age on Standardized, Uptake Value-Hounsfield Unit Values of Male Genitourinery Structures In F-18 FDG PET/CT.

6. Testosterone reduces knee passive range of motion and expression of relaxin receptor isoforms via 5α-dihydrotestosterone and androgen receptor binding.