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8 in total

1. Apicomplexan parasite, Eimeria falciformis, co-opts host tryptophan catabolism for life cycle progression in mouse.

Authors: Manuela Schmid; Maik J Lehmann; Richard Lucius; Nishith Gupta
Journal: J Biol Chem Date: 2012-04-25 Impact factor: 5.157

2. Lipid analysis of Eimeria sporozoites reveals exclusive phospholipids, a phylogenetic mosaic of endogenous synthesis, and a host-independent lifestyle.

Authors: Pengfei Kong; Maik J Lehmann; J Bernd Helms; Jos F Brouwers; Nishith Gupta
Journal: Cell Discov Date: 2018-05-22 Impact factor: 10.849

3. T-cell alpha beta + and gamma delta + deficient mice display abnormal but distinct phenotypes toward a natural, widespread infection of the intestinal epithelium.

Authors: S J Roberts; A L Smith; A B West; L Wen; R C Findly; M J Owen; A C Hayday
Journal: Proc Natl Acad Sci U S A Date: 1996-10-15 Impact factor: 11.205

4. Immunization against experimental coccidiosis produces contrasting results in inbred mice of differing susceptibility to infection.

Authors: M E Rose; P Hesketh; D Wakelin
Journal: Infect Immun Date: 1994-02 Impact factor: 3.441

5. Eimeria falciformis infection of the mouse caecum identifies opposing roles of IFNγ-regulated host pathways for the parasite development.

Authors: Manuela Schmid; Emanuel Heitlinger; Simone Spork; Hans-Joachim Mollenkopf; Richard Lucius; Nishith Gupta
Journal: Mucosal Immunol Date: 2013-12-25 Impact factor: 7.313

Mouse strain variation and effects of oocyst dose in infection of mice with Eimeria falciformis, a coccidian parasite of the large intestine.

1. Apicomplexan parasite, Eimeria falciformis, co-opts host tryptophan catabolism for life cycle progression in mouse.

2. Lipid analysis of Eimeria sporozoites reveals exclusive phospholipids, a phylogenetic mosaic of endogenous synthesis, and a host-independent lifestyle.

3. T-cell alpha beta + and gamma delta + deficient mice display abnormal but distinct phenotypes toward a natural, widespread infection of the intestinal epithelium.

4. Immunization against experimental coccidiosis produces contrasting results in inbred mice of differing susceptibility to infection.

5. Eimeria falciformis infection of the mouse caecum identifies opposing roles of IFNγ-regulated host pathways for the parasite development.

6. The role of natural killer cells in resistance to coccidiosis: investigations in a murine model.

7. Responses to vaccination in strains of mice that differ in susceptibility to coccidiosis.

8. Toxoplasma and Eimeria co-opt the host cFos expression for intracellular development in mammalian cells.