Literature DB >> 3860585

Adenosine triphosphate degradation in specific disease.

I H Fox.   

Abstract

Two hypotheses concerning adenosine triphosphate (ATP) degradation are examined: The metabolic basis of specific disorders involves ATP degradation, and uric acid and its precursors are measurable body fluid markers of ATP degradation. These hypotheses are examined by discussing human models of ATP degradation, methods for measurement of ATP degradation, and disorders of ATP degradation.

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Year:  1985        PMID: 3860585

Source DB:  PubMed          Journal:  J Lab Clin Med        ISSN: 0022-2143


  5 in total

1.  Relation between creatinine and uric acid excretion.

Authors:  Y Nishida
Journal:  Ann Rheum Dis       Date:  1992-01       Impact factor: 19.103

Review 2.  Clinical and biochemical aspects of uric acid overproduction.

Authors:  J García Puig; F A Mateos
Journal:  Pharm World Sci       Date:  1994-04-15

3.  Hereditary xanthinuria. Evidence for enhanced hypoxanthine salvage.

Authors:  F A Mateos; J G Puig; M L Jiménez; I H Fox
Journal:  J Clin Invest       Date:  1987-03       Impact factor: 14.808

4.  Adenosine triphosphate turnover in humans. Decreased degradation during relative hyperphosphatemia.

Authors:  M A Johnson; K Tekkanat; S P Schmaltz; I H Fox
Journal:  J Clin Invest       Date:  1989-09       Impact factor: 14.808

5.  Skeletal muscle weakness is related to insulin resistance in patients with chronic heart failure.

Authors:  Wolfram Doehner; Guelistan Turhan; Francisco Leyva; Mathias Rauchhaus; Anja Sandek; Ewa A Jankowska; Stephan von Haehling; Stefan D Anker
Journal:  ESC Heart Fail       Date:  2015-04-30
  5 in total

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