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Literature DB >> 377995

Location of brown recluse venom attachment sites on human erythrocytes by the firritin-labeled antibody technique.

J M Futrell, P N Morgan, S P Su, S I Roth.

Abstract

Brown recluse spider (loxosceles reclusa) venom has been demonstrated by a ferritin-labeled antibody technique to attach to human erythrocyte cell membranes. The number of individual attachment sites per cell is proportional to the concentration of the venom used to sensitize the erythrocytes. Structural changes in the red cell membrane are associated with the venom attachment. These sites may be related to the red cell hemolysis which sometimes occurs in the human as a result of the spider bite.

Entities: Disease Species

Mesh：

Substances：

Year: 1979 PMID： 377995 PMCID： PMC2042310

Source DB: PubMed Journal: Am J Pathol ISSN： 0002-9440 Impact factor: 4.307

10 in total

1. The gangrenous bite of the Brown Recluse spider in Arkansas.

Authors: C J DILLAHA; G T JANSEN; W M HONEYCUTT; C R HAYDEN
Journal: J Ark Med Soc Date: 1963-08

2. Hemolytic anemia from brown spider bite, necrotic arachnidism.

Authors: J F NICHOLSON; B H NICHOLSON
Journal: J Okla State Med Assoc Date: 1962-06

3. The role of polymorphonuclear leukocytes in the lesion caused by the venom of the brown spider, Loxosceles reclusa.

Authors: C W Smith; D W Micks
Journal: Lab Invest Date: 1970-01 Impact factor: 5.662

4. Variation in severity of loxoscelism.

Authors: H B Reed; R H Hackman; F M Fesmire
Journal: J Tenn Med Assoc Date: 1968-11

5. Hemolysis, renal failure and death, presumed secondary to bite of brown recluse spider.

Authors: E H Taylor; W F Denny
Journal: South Med J Date: 1966-10 Impact factor: 0.954

6. Identification and neutralization of biological activities associated with venom from the brown recluse spider, Loxosceles reclusa.

Authors: J M Futrell; P N Morgan
Journal: Am J Trop Med Hyg Date: 1977-11 Impact factor: 2.345

1 in total

1. Brown Recluse spider bite mediated hemolysis: clinical features, a possible role for complement inhibitor therapy, and reduced RBC surface glycophorin A as a potential biomarker of venom exposure.

Authors: Eric A Gehrie; Hui Nian; Pampee P Young
Journal: PLoS One Date: 2013-09-27 Impact factor: 3.240

1 in total

Location of brown recluse venom attachment sites on human erythrocytes by the firritin-labeled antibody technique.

1. The gangrenous bite of the Brown Recluse spider in Arkansas.

2. Hemolytic anemia from brown spider bite, necrotic arachnidism.

3. The role of polymorphonuclear leukocytes in the lesion caused by the venom of the brown spider, Loxosceles reclusa.

4. Variation in severity of loxoscelism.

5. Hemolysis, renal failure and death, presumed secondary to bite of brown recluse spider.

6. Identification and neutralization of biological activities associated with venom from the brown recluse spider, Loxosceles reclusa.

7. Lysis of human erythrocytes by venom from the brown recluse spider, Loxosceles reclusa.

8. An inhibitor of complement in the venom of the brown recluse spider, Loxosceles reclusa.

9. Preliminary studies on venom from the brown recluse spider Loxosceles reclusa.

10. Improvements in epoxy resin embedding methods.

1. Brown Recluse spider bite mediated hemolysis: clinical features, a possible role for complement inhibitor therapy, and reduced RBC surface glycophorin A as a potential biomarker of venom exposure.