Literature DB >> 36239974

Optic Nerve Head Myelin-Related Protein, GFAP, and Iba1 Alterations in Non-Human Primates With Early to Moderate Experimental Glaucoma.

Priya Chaudhary1,2, Cheri Stowell1,2, Juan Reynaud1,2, Stuart K Gardiner2, Hongli Yang1,2, Galen Williams1,2, Imee Williams1,2, Nicholas Marsh-Armstrong3, Claude F Burgoyne1,2.   

Abstract

Purpose: The purpose of this study was to test if optic nerve head (ONH) myelin basic protein (MBP), 2',3'-cyclic nucleotide 3'-phosphodiesterase (CNPase), glial fibrillary acidic protein (GFAP), and ionized calcium binding adaptor molecule 1 (Iba1) proteins are altered in non-human primate (NHP) early/moderate experimental glaucoma (EG).
Methods: Following paraformaldehyde perfusion, control and EG eye ONH tissues from four NHPs were paraffin embedded and serially (5 µm) vertically sectioned. Anti-MBP, CNPase, GFAP, Iba1, and nuclear dye-stained sections were imaged using sub-saturating light intensities. Whole-section images were segmented creating anatomically consistent laminar (L) and retrolaminar (RL) regions/sub-regions. EG versus control eye intensity/pixel-cluster density data within L and two RL regions (RL1 [1-250 µm]/RL2 [251-500 µm] from L) were compared using random effects models within the statistical program "R."
Results: EG eye retinal nerve fiber loss ranged from 0% to 20%. EG eyes' MBP and CNPase intensity were decreased within the RL1 (MBP = 31.4%, P < 0.001; CNPase =62.3%, P < 0.001) and RL2 (MBP = 19.6%, P < 0.001; CNPase = 56.1%, P = 0.0004) regions. EG eye GFAP intensity was decreased in the L (41.6%, P < 0.001) and RL regions (26.7% for RL1, and 28.4% for RL2, both P < 0.001). Iba1+ and NucBlue pixel-cluster density were increased in the laminar (28.2%, P = 0.03 and 16.6%, P = 0.008) and both RL regions (RL1 = 37.3%, P = 0.01 and 23.7%, P = 0.0002; RL2 = 53.7%, P = 0.002 and 33.2%, P < 0.001). Conclusions: Retrolaminar myelin disruption occurs early in NHP EG and may be accompanied by laminar and retrolaminar decreases in astrocyte process labeling and increases in microglial/ macrophage density. The mechanistic and therapeutic implications of these findings warrant further study.

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Year:  2022        PMID: 36239974      PMCID: PMC9586137          DOI: 10.1167/iovs.63.11.9

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.925


  91 in total

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2.  Radiation treatment inhibits monocyte entry into the optic nerve head and prevents neuronal damage in a mouse model of glaucoma.

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4.  Phagocytosis in the rat optic nerve following Wallerian degeneration.

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7.  Early astrocyte redistribution in the optic nerve precedes axonopathy in the DBA/2J mouse model of glaucoma.

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8.  Ocular hypertension suppresses homeostatic gene expression in optic nerve head microglia of DBA/2 J mice.

Authors:  James R Tribble; Jeffrey M Harder; Pete A Williams; Simon W M John
Journal:  Mol Brain       Date:  2020-05-25       Impact factor: 4.041

Review 9.  Modulation of the Immune System for the Treatment of Glaucoma.

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Review 10.  The Role of Microglia in Retinal Neurodegeneration: Alzheimer's Disease, Parkinson, and Glaucoma.

Authors:  Ana I Ramirez; Rosa de Hoz; Elena Salobrar-Garcia; Juan J Salazar; Blanca Rojas; Daniel Ajoy; Inés López-Cuenca; Pilar Rojas; Alberto Triviño; José M Ramírez
Journal:  Front Aging Neurosci       Date:  2017-07-06       Impact factor: 5.750

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