Literature DB >> 36087241

Quantitative T2* MRI for bone marrow iron overload: normal reference values and assessment in thalassemia major patients.

Antonella Meloni1,2, Laura Pistoia1, Gennaro Restaino3, Massimiliano Missere3, Vincenzo Positano1,2, Anna Spasiano4, Tommaso Casini5, Antonella Cossu6, Liana Cuccia7, Antonella Massa8, Francesco Massei9, Filippo Cademartiri10.   

Abstract

PURPOSE: We evaluated the feasibility and reproducibility of bone marrow T2* values and established the lower limit of normal in a cohort of healthy subjects. We investigated the clinical correlates of bone marrow T2* values in patients with thalassemia major (TM).
MATERIAL AND METHODS: Thirty healthy subjects and 274 consecutive TM patients (38.96 ± 8.49 years, 151 females) underwent MRI at 1.5T. An axial slice in the upper abdomen was acquired by a T2* gradient-echo multiecho sequence and the T2* value was calculated in a circular region of interest defined in the visible body of the first or second lumbar vertebra. In patients, also liver and heart T2* values were assessed.
RESULTS: In healthy subjects bone marrow T2* values were independent of age and gender. The lower limit of normal for bone marrow T2* was 13 ms. In both healthy subjects and 30 randomly selected patients, the coefficient of variation for inter-operator-reproducibility was < 10%. TM patients exhibited significantly lower bone marrow T2* values than healthy subjects (7.47 ± 5.18 ms vs. 17.08 ± 1.89 ms; p < 0.0001). A pathological bone marrow T2* was detected in 82.8% of TM patients. In TM, the female sex was associated with reduced bone marrow T2* values. Bone marrow T2* values were inversely correlated with mean serum ferritin levels (R = -0.431; P < 0.0001) and hepatic iron load (R = - 0.215; P < 0.0001). A serum ferritin level > 536 ng/ml predicted the presence of a pathological bone marrow T2*. A positive correlation was found between bone marrow and heart T2* values (R = 0.143; P = 0.018). A normal bone marrow T2* showed a negative predictive value of 100% for cardiac iron.
CONCLUSION: Bone marrow T2* measurements can be easily obtained using the same sequences acquired for liver iron quantification and may bring new insights into the pathophysiology of iron deposition; hence, they should be incorporated into clinical practice.
© 2022. Italian Society of Medical Radiology.

Entities:  

Keywords:  Bone marrow; Iron overload; Lower limit of normal; Magnetic resonance imaging; Thalassemia major

Year:  2022        PMID: 36087241     DOI: 10.1007/s11547-022-01554-w

Source DB:  PubMed          Journal:  Radiol Med        ISSN: 0033-8362            Impact factor:   6.313


  17 in total

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Authors:  D J Weatherall; A B Provan
Journal:  Lancet       Date:  2000-04-01       Impact factor: 79.321

2.  Increased bone marrow iron stores is an independent risk factor for invasive aspergillosis in patients with high-risk hematologic malignancies and recipients of allogeneic hematopoietic stem cell transplantation.

Authors:  Dimitrios P Kontoyiannis; Georgios Chamilos; Russell E Lewis; Sergio Giralt; Jorge Cortes; Issam I Raad; John T Manning; Xin Han
Journal:  Cancer       Date:  2007-09-15       Impact factor: 6.860

3.  Dual-energy CT for liver iron quantification in patients with haematological disorders.

Authors:  Sebastian Werner; Bernhard Krauss; Ulrike Haberland; Malte Bongers; Uwe Starke; Tamam Bakchoul; Sigrid Enkel; Konstantin Nikolaou; Marius Horger
Journal:  Eur Radiol       Date:  2018-11-07       Impact factor: 5.315

4.  Renal iron deposition by magnetic resonance imaging in pediatric β-thalassemia major patients: Relation to renal biomarkers, total body iron and chelation therapy.

Authors:  Mohsen Saleh ElAlfy; Nayera Hazaa Khalil Elsherif; Fatma Soliman Elsayed Ebeid; Eman Abdel Rahman Ismail; Khaled Aboulfotouh Ahmed; Yasser Wagih Darwish; Ahmed Samir Ibrahim; Islam Reda Farag Elghamry; Nermeen Adel Shokrey; Dunia Naser Alajeil
Journal:  Eur J Radiol       Date:  2018-04-09       Impact factor: 3.528

5.  Improvement in hematopoiesis after iron chelation therapy with deferasirox in patients with aplastic anemia.

Authors:  Sung-Eun Lee; Seung-Ah Yahng; Byung-Sik Cho; Ki-Sung Eom; Yoo-Jin Kim; Seok Lee; Chang-Ki Min; Hee-Je Kim; Seok-Goo Cho; Dong-Wook Kim; Woo-Sung Min; Chong-Won Park; Jong Wook Lee
Journal:  Acta Haematol       Date:  2012-11-15       Impact factor: 2.195

6.  Internal distribution of excess iron and sources of serum ferritin in patients with thalassemia.

Authors:  M Cazzola; C Borgna-Pignatti; P de Stefano; G Bergamaschi; I G Bongo; L Dezza; F Avato
Journal:  Scand J Haematol       Date:  1983-04

7.  Iron overload causes osteoporosis in thalassemia major patients through interaction with transient receptor potential vanilloid type 1 (TRPV1) channels.

Authors:  Francesca Rossi; Silverio Perrotta; Giulia Bellini; Livio Luongo; Chiara Tortora; Dario Siniscalco; Matteo Francese; Marco Torella; Bruno Nobili; Vincenzo Di Marzo; Sabatino Maione
Journal:  Haematologica       Date:  2014-09-12       Impact factor: 9.941

8.  Secondary solid cancer following hematopoietic cell transplantation in patients with thalassemia major.

Authors:  S Santarone; A Pepe; A Meloni; A Natale; L Pistoia; P Olioso; G Papalinetti; L Cuccia; A Spasiano; R Lisi; M Di Ianni; T Bonfini; P Accorsi; S Salvadori; F Papola; S Angelini; P Di Bartolomeo
Journal:  Bone Marrow Transplant       Date:  2017-10-09       Impact factor: 5.483

9.  The bone marrow hematopoietic microenvironment is impaired in iron-overloaded mice.

Authors:  Hiroshi Okabe; Takahiro Suzuki; Eisuke Uehara; Masuzu Ueda; Tadashi Nagai; Keiya Ozawa
Journal:  Eur J Haematol       Date:  2014-03-28       Impact factor: 2.997

10.  ROS-mediated iron overload injures the hematopoiesis of bone marrow by damaging hematopoietic stem/progenitor cells in mice.

Authors:  Xiao Chai; Deguan Li; Xiaoli Cao; Yuchen Zhang; Juan Mu; Wenyi Lu; Xia Xiao; Chengcheng Li; Juanxia Meng; Jie Chen; Qing Li; Jishi Wang; Aimin Meng; Mingfeng Zhao
Journal:  Sci Rep       Date:  2015-05-13       Impact factor: 4.379

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