Literature DB >> 35996042

Variable phenotypes of individual and family monogenic cases with hyperinsulinism and diabetes: a systematic review.

Kevin Perge1,2, Marc Nicolino3,4.   

Abstract

Maturity-Onset Diabetes of the Youth (MODY) diabetes remains commonly misdiagnosed. A monogenic form should be suspected in individuals presenting hyperinsulinemic hypoglycemia (HH) associated with, either later development of MODY (hypoglycemia-remission-diabetes sequence), or with first/second-degree family history of diabetes. Herein, we aimed to describe this individual or family monogenic association between HH and diabetes, and identify potential genotype-phenotype correlations. We conducted a systematic review of 26 studies, including a total of 67 patients with this association resulting from variants in GCK (n = 5 cases), ABCC8 (n = 29), HNF1A (n = 5), or HNF4A (n = 28). A family history of hypoglycemia and/or diabetes was present in 91% of cases (61/67). Median age at first hypoglycemia was 24 h after birth. Diazoxide was initiated in 46 children (46/67-69%); responsiveness was found in 91% (42/46). Median HH duration was three years (1 day-25 years). Twenty-three patients (23/67-34%) later developed diabetes (median age: 13 years; range: 8-48); more frequently in those untreated with diazoxide. This association was most commonly inherited in an autosomal dominant manner (43/48-90%). Some genes were associated with less severe initial hypoglycemia (HNF1A), shorter duration of HH (HNF4A), and more maternal (ABCC8) or paternal (HNF4A) transmission. This study illustrates that the same genotype can give a biphasic phenotype in the same person or a reverse phenotype in the same family. Wider awareness of this association is necessary in pediatrics to establish annual monitoring of patients who have presented HH, and during maternity to screen diabetes and optimize genetic counseling and management of pregnancy, childbirth, and the newborn.PROSPERO registration: CRD42020178265.
© 2022. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.

Entities:  

Keywords:  Congenital hyperinsulinism; Diabetes mellitus; Genetics; Hypoglycemia; MODY diabetes; Monogenic diabetes

Mesh:

Substances:

Year:  2022        PMID: 35996042     DOI: 10.1007/s11154-022-09749-2

Source DB:  PubMed          Journal:  Rev Endocr Metab Disord        ISSN: 1389-9155            Impact factor:   9.306


  56 in total

1.  Dominantly inherited hyperinsulinism caused by a mutation in the sulfonylurea receptor type 1.

Authors:  H Huopio; F Reimann; R Ashfield; J Komulainen; H L Lenko; J Rahier; I Vauhkonen; J Kere; M Laakso; F Ashcroft; T Otonkoski
Journal:  J Clin Invest       Date:  2000-10       Impact factor: 14.808

2.  Familial leucine-sensitive hypoglycemia of infancy due to a dominant mutation of the beta-cell sulfonylurea receptor.

Authors:  Sheela N Magge; Show-Ling Shyng; Courtney MacMullen; Linda Steinkrauss; Arupa Ganguly; Lorraine E L Katz; Charles A Stanley
Journal:  J Clin Endocrinol Metab       Date:  2004-09       Impact factor: 5.958

3.  Maturity-onset diabetes of the young (MODY): how many cases are we missing?

Authors:  B M Shields; S Hicks; M H Shepherd; K Colclough; A T Hattersley; S Ellard
Journal:  Diabetologia       Date:  2010-05-25       Impact factor: 10.122

4.  The PRISMA statement for reporting systematic reviews and meta-analyses of studies that evaluate health care interventions: explanation and elaboration.

Authors:  Alessandro Liberati; Douglas G Altman; Jennifer Tetzlaff; Cynthia Mulrow; Peter C Gøtzsche; John P A Ioannidis; Mike Clarke; P J Devereaux; Jos Kleijnen; David Moher
Journal:  J Clin Epidemiol       Date:  2009-07-23       Impact factor: 6.437

5.  Prevalence, characteristics and clinical diagnosis of maturity onset diabetes of the young due to mutations in HNF1A, HNF4A, and glucokinase: results from the SEARCH for Diabetes in Youth.

Authors:  Catherine Pihoker; Lisa K Gilliam; Sian Ellard; Dana Dabelea; Cralen Davis; Lawrence M Dolan; Carla J Greenbaum; Giuseppina Imperatore; Jean M Lawrence; Santica M Marcovina; Elizabeth Mayer-Davis; Beatriz L Rodriguez; Andrea K Steck; Desmond E Williams; Andrew T Hattersley
Journal:  J Clin Endocrinol Metab       Date:  2013-06-14       Impact factor: 5.958

6.  Glucose intolerance and diabetes are observed in the long-term follow-up of nonpancreatectomized patients with persistent hyperinsulinemic hypoglycemia of infancy due to mutations in the ABCC8 gene.

Authors:  Miquel Gussinyer; María Clemente; Rocio Cebrián; Diego Yeste; Marian Albisu; Antonio Carrascosa
Journal:  Diabetes Care       Date:  2008-03-13       Impact factor: 19.112

7.  Clinical characteristics and biochemical mechanisms of congenital hyperinsulinism associated with dominant KATP channel mutations.

Authors:  Sara E Pinney; Courtney MacMullen; Susan Becker; Yu-Wen Lin; Cheryl Hanna; Paul Thornton; Arupa Ganguly; Show-Ling Shyng; Charles A Stanley
Journal:  J Clin Invest       Date:  2008-08       Impact factor: 14.808

8.  Hyperinsulinemic hypoglycemia evolving to gestational diabetes and diabetes mellitus in a family carrying the inactivating ABCC8 E1506K mutation.

Authors:  Teresa C Vieira; Carla S Bergamin; Lucimary C Gurgel; Regina S Moisés
Journal:  Pediatr Diabetes       Date:  2010-11       Impact factor: 4.866

9.  Macrosomia and hyperinsulinaemic hypoglycaemia in patients with heterozygous mutations in the HNF4A gene.

Authors:  Ewan R Pearson; Sylvia F Boj; Anna M Steele; Timothy Barrett; Karen Stals; Julian P Shield; Sian Ellard; Jorge Ferrer; Andrew T Hattersley
Journal:  PLoS Med       Date:  2007-04       Impact factor: 11.069

10.  Novel de novo mutation in sulfonylurea receptor 1 presenting as hyperinsulinism in infancy followed by overt diabetes in early adolescence.

Authors:  Maha Abdulhadi-Atwan; Jeremy Bushman; Jeremy Bushmann; Sharona Tornovsky-Babaey; Avital Perry; Abdulsalam Abu-Libdeh; Benjamin Glaser; Show-Ling Shyng; David H Zangen
Journal:  Diabetes       Date:  2008-04-04       Impact factor: 9.461

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