Correction to: Long noncoding RNA BCRP3 stimulates VPS34 and autophagy activities to promote protein homeostasis and cell survival.

Correction: Journal of Biomedical Science (2022) 29:30 https://doi.org/10.1186/s12929-022-00815-0

After the publication of this article [1], we noted that the GAPDH blot of the Fig. 7C right panel is incorrect. The corrected Fig. 7C is included below.

Fig. 7

BCRP3 defciency in proteotoxicity leads to the accumulation of proteins involving in growth inhibition, cell death, and TGF-β/Smad2 signaling. A Venn diagram showing the numbers of enriched proteins after BCRP3 knockdown together with or without 10 µM MG132 treatment for 12 h. B GO enrichment analysis of the 134 proteins shown in (A). Selective enriched GO terms are shown by the order of fold enrichment (bottom to top). C Western blot analysis of indicated proteins in control or BCRP3-defcient HeLa cells treated with 10 µM MG132 together for 12 h together with or without 200 nM baflomycin A1 for 2 h. D Control or BCRP3-defcient HeLa cells were transfected with 4 × SBE-Luc reporter construct, treated with 10 µM MG132 for 12 h and analyzed for luciferase activity. E qRT-PCR analysis of relative DAPK1, p15, and p21 levels in control or BCRP3-defcient HeLa cells treated with 10 µM MG132 for 12 h. Data in (D), (E) are means ± SD from three independent experiments. P values are determined by one-way ANOVA with Tukey’s post hoc test, *P < 0.001