Literature DB >> 35953577

Evaluation of optical coherence tomography findings and visual evoked potentials in Charcot-Marie-Tooth disease.

Aysin Tuba Kaplan1, Sibel Oskan Yalcin2, Safiye Gunes Sager3, Ayberk Türkyılmaz4, Rahşan İnan5.   

Abstract

PURPOSE: To evaluate the spectral-domain optical coherence tomography (SD-OCT) findings and pattern visual evoked potential (VEP) in Charcot-Marie-Tooth (CMT) disease.
METHODS: Seventeen patients with CMT disease and 17 control subjects were included in the study. The patients were divided into two groups according to conduction velocity and inheritance pattern as demyelinating type (CMT 1) and axonal type (CMT 2). The average retinal nerve fiber layer (RNFL) thickness, RNFL thicknesses of all quadrants, and thicknesses of the ganglion cell layer complex (GCC) were measured using SD-OCT. Pattern VEP recordings were evaluated in both groups.
RESULTS: The average and four quadrants of RNFL thicknesses, and superior and inferior GCC thicknesses were significantly thinner in the CMT patients compared with healthy individuals, but there were no statistically significant differences between the CMT groups. There was a significant positive correlation between age and all RNFL and GCC thicknesses in the CMT 2 group and between age and RNFL thickness of the temporal quadrant in the CMT 1 group. P100 latencies were significantly delayed in the CMT groups compared with controls, and there were no significant differences in P100 latencies between the CMT groups (p < 0.001). VEP amplitudes were in normal ranges in the CMT groups.
CONCLUSION: This study showed that RNFL and GCC thicknesses were significantly reduced and VEP latencies were prolonged in patients with CMT with normal clinical examinations. Our results suggest that optic nerves may be affected more frequently in patients with CMT that is detected in clinical examinations.
© 2022. The Author(s), under exclusive licence to Springer Nature B.V.

Entities:  

Keywords:  Axonal type; Demyelinating; Ganglion cell; Latency; Retinal nerve fiber layer

Year:  2022        PMID: 35953577     DOI: 10.1007/s10792-022-02452-w

Source DB:  PubMed          Journal:  Int Ophthalmol        ISSN: 0165-5701            Impact factor:   2.029


  3 in total

1.  Axonal neuropathy with optic atrophy is caused by mutations in mitofusin 2.

Authors:  Stephan Züchner; Peter De Jonghe; Albena Jordanova; Kristl G Claeys; Velina Guergueltcheva; Sylvia Cherninkova; Steven R Hamilton; Greg Van Stavern; Karen M Krajewski; Jeffery Stajich; Ivajlo Tournev; Kristien Verhoeven; Christine T Langerhorst; Marianne de Visser; Frank Baas; Thomas Bird; Vincent Timmerman; Michael Shy; Jeffery M Vance
Journal:  Ann Neurol       Date:  2006-02       Impact factor: 10.422

2.  Characterization of Charcot-Marie-Tooth optic neuropathy.

Authors:  Benjamin Botsford; Laurel N Vuong; Thomas R Hedges; Carlos E Mendoza-Santiesteban
Journal:  J Neurol       Date:  2017-10-23       Impact factor: 4.849

3.  Diagnostic genetic testing for patients with bilateral optic neuropathy and comparison of clinical features according to OPA1 mutation status.

Authors:  Eric D Gaier; Katherine Boudreault; Isao Nakata; Maria Janessian; Philip Skidd; Elizabeth DelBono; Keri F Allen; Louis R Pasquale; Emily Place; Dean M Cestari; Rebecca C Stacy; Joseph F Rizzo; Janey L Wiggs
Journal:  Mol Vis       Date:  2017-08-10       Impact factor: 2.367

  3 in total

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