Literature DB >> 35798978

A quantitative in vivo imaging platform for tracking pathological tau depositions and resultant neuronal death in a mouse model.

Taeko Kimura1, Maiko Ono1, Chie Seki2, Kazuaki Sampei1, Masafumi Shimojo1, Kazunori Kawamura1, Ming-Rong Zhang1, Naruhiko Sahara1, Yuhei Takado3, Makoto Higuchi1.   

Abstract

PURPOSE: Depositions of tau fibrils are implicated in diverse neurodegenerative disorders, including Alzheimer's disease, and precise assessments of tau pathologies and their impacts on neuronal survival are crucial for pursuing the neurodegenerative tau pathogenesis with and without potential therapies. We aimed to establish an in vivo imaging system to quantify tau accumulations with positron emission tomography (PET) and brain atrophy with volumetric MRI in rTg4510 transgenic mice modeling neurodegenerative tauopathies.
METHODS: A total of 91 rTg4510 and non-transgenic control mice underwent PET with a tau radiotracer, 18F-PM-PBB3, and MRI at various ages (1.8-12.3 months). Using the cerebellum as reference, the radiotracer binding in target regions was estimated as standardized uptake value ratio (SUVR) and distribution volume ratio (DVR). Histopathological staining of brain sections derived from scanned animals was also conducted to investigate the imaging-neuropathology correlations.
RESULTS: 18F-PM-PBB3 SUVR at 40-60 min in the neocortex, hippocampus, and striatum of rTg4510 mice agreed with DVR, became significantly different from control values around 4-5 months of age, and progressively and negatively correlated with age and local volumes, respectively. Neocortical SUVR also correlated with the abundance of tau inclusions labeled with PM-PBB3 fluorescence, Gallyas-Braak silver impregnation, and anti-phospho-tau antibodies in postmortem assays. The in vivo and ex vivo 18F-PM-PBB3 binding was blocked by non-radioactive PM-PBB3. 18F-PM-PBB3 yielded a 1.6-fold greater dynamic range for tau imaging than its ancestor, 11C-PBB3.
CONCLUSION: Our imaging platform has enabled the quantification of tau depositions and consequent neuronal loss and is potentially applicable to the evaluation of candidate anti-tau and neuroprotective drugs.
© 2022. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.

Entities:  

Keywords:  18F-PM-PBB3 (APN-1607; Florzolotau); Small-animal PET; Tauopathy; Volumetric MR imaging; rTg4510 mouse

Year:  2022        PMID: 35798978     DOI: 10.1007/s00259-022-05898-3

Source DB:  PubMed          Journal:  Eur J Nucl Med Mol Imaging        ISSN: 1619-7070            Impact factor:   9.236


  21 in total

Review 1.  Neurodegenerative tauopathies.

Authors:  V M Lee; M Goedert; J Q Trojanowski
Journal:  Annu Rev Neurosci       Date:  2001       Impact factor: 12.449

Review 2.  Mutations causing neurodegenerative tauopathies.

Authors:  Michel Goedert; Ross Jakes
Journal:  Biochim Biophys Acta       Date:  2005-01-03

3.  Overview of Transgenic Mouse Models for Alzheimer's Disease.

Authors:  Ariana Myers; Paul McGonigle
Journal:  Curr Protoc Neurosci       Date:  2019-09

4.  Hsp90 activator Aha1 drives production of pathological tau aggregates.

Authors:  Lindsey B Shelton; Jeremy D Baker; Dali Zheng; Leia E Sullivan; Parth K Solanki; Jack M Webster; Zheying Sun; Jonathan J Sabbagh; Bryce A Nordhues; John Koren; Suman Ghosh; Brian S J Blagg; Laura J Blair; Chad A Dickey
Journal:  Proc Natl Acad Sci U S A       Date:  2017-08-21       Impact factor: 11.205

5.  High-Contrast In Vivo Imaging of Tau Pathologies in Alzheimer's and Non-Alzheimer's Disease Tauopathies.

Authors:  Kenji Tagai; Maiko Ono; Manabu Kubota; Soichiro Kitamura; Keisuke Takahata; Chie Seki; Yuhei Takado; Hitoshi Shinotoh; Yasunori Sano; Yasuharu Yamamoto; Kiwamu Matsuoka; Hiroyuki Takuwa; Masafumi Shimojo; Manami Takahashi; Kazunori Kawamura; Tatsuya Kikuchi; Maki Okada; Haruhiko Akiyama; Hisaomi Suzuki; Mitsumoto Onaya; Takahiro Takeda; Kimihito Arai; Nobutaka Arai; Nobuyuki Araki; Yuko Saito; John Q Trojanowski; Virginia M Y Lee; Sushil K Mishra; Yoshiki Yamaguchi; Yasuyuki Kimura; Masanori Ichise; Yutaka Tomita; Ming-Rong Zhang; Tetsuya Suhara; Masahiro Shigeta; Naruhiko Sahara; Makoto Higuchi; Hitoshi Shimada
Journal:  Neuron       Date:  2020-10-29       Impact factor: 17.173

Review 6.  In-vivo visualization of key molecular processes involved in Alzheimer's disease pathogenesis: Insights from neuroimaging research in humans and rodent models.

Authors:  Makoto Higuchi; Jun Maeda; Bin Ji; Masahiro Maruyama; Takashi Okauchi; Masaki Tokunaga; Maiko Ono; Tetsuya Suhara
Journal:  Biochim Biophys Acta       Date:  2010-01-12

Review 7.  Development of disease-modifying drugs for frontotemporal dementia spectrum disorders.

Authors:  Francesco Panza; Bruno P Imbimbo; Madia Lozupone; Davide Seripa; Antonio Daniele; Mark Watling; Gianluigi Giannelli
Journal:  Nat Rev Neurol       Date:  2020-03-23       Impact factor: 42.937

Review 8.  Tau-based therapies in neurodegeneration: opportunities and challenges.

Authors:  Chuanzhou Li; Jürgen Götz
Journal:  Nat Rev Drug Discov       Date:  2017-10-06       Impact factor: 84.694

Review 9.  A review: treatment of Alzheimer's disease discovered in repurposed agents.

Authors:  Brian S Appleby; Dimitrios Nacopoulos; Nicholas Milano; Kate Zhong; Jeffrey L Cummings
Journal:  Dement Geriatr Cogn Disord       Date:  2013-01-09       Impact factor: 2.959

10.  Assessment of Tau Pathology as Measured by 18F-THK5317 and 18F-Flortaucipir PET and Their Relation to Brain Atrophy and Cognition in Alzheimer's Disease.

Authors:  Elisa Colato; Konstantinos Chiotis; Daniel Ferreira; Mariam S Mazrina; Laetitia Lemoine; Rosaleena Mohanty; Eric Westman; Agneta Nordberg; Elena Rodriguez-Vieitez
Journal:  J Alzheimers Dis       Date:  2021       Impact factor: 4.472

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.