Literature DB >> 35674212

Localized islet nuclear enlargement hyperinsulinism (LINE-HI) due to ABCC8 and GCK mosaic mutations.

Kara E Boodhansingh1, Zhongying Yang2, Changhong Li1, Pan Chen1, Katherine Lord1,3, Susan A Becker4, Lisa J States4,5, N Scott Adzick6, Tricia Bhatti7,8, Show-Ling Shyng2, Arupa Ganguly9, Charles A Stanley1,3, Diva D De Leon1,3.   

Abstract

Objective: Congenital hyperinsulinism (HI) is the most common cause of persistent hypoglycemia in children. In addition to typical focal or diffuse HI, some cases with diazoxide-unresponsive congenital HI have atypical pancreatic histology termed Localized Islet Nuclear Enlargement (LINE) or mosaic HI, characterized by histologic features similar to diffuse HI, but confined to only a region of pancreas. Our objective was to characterize the phenotype and genotype of children with LINE-HI. Design: The phenotype and genotype features of 12 children with pancreatic histology consistent with LINE-HI were examined.
Methods: We compiled clinical features of 12 children with LINE-HI and performed next-generation sequencing on specimens of pancreas from eight of these children to look for mosaic mutations in genes known to be associated with diazoxide-unresponsive HI (ABCC8, KCNJ11, and GCK).
Results: Children with LINE-HI had lower birth weights and later ages of presentation compared to children with typical focal or diffuse HI. Partial pancreatectomy in LINE-HI cases resulted in euglycemia in 75% of cases; no cases have developed diabetes. Low-level mosaic mutations were identified in the pancreas of six cases with LINE-HI (three in ABCC8, three in GCK). Expression studies confirmed that all novel mutations were pathogenic.
Conclusion: These results indicate that post-zygotic low-level mosaic mutations of known HI genes are responsible for some cases of LINE-HI that lack an identifiable germ-line mutation and that partial pancreatectomy may be curative for these cases.

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Year:  2022        PMID: 35674212      PMCID: PMC9339501          DOI: 10.1530/EJE-21-1095

Source DB:  PubMed          Journal:  Eur J Endocrinol        ISSN: 0804-4643            Impact factor:   6.558


  56 in total

1.  Mutational analysis of allosteric activation and inhibition of glucokinase.

Authors:  Bogumil Zelent; Stella Odili; Carol Buettger; Dorothy K Zelent; Pan Chen; Deborah Fenner; Joseph Bass; Charles Stanley; Monique Laberge; Jane M Vanderkooi; Ramakanth Sarabu; Joseph Grimsby; Franz M Matschinsky
Journal:  Biochem J       Date:  2011-12-01       Impact factor: 3.857

2.  Hyperinsulinism and hyperammonemia in infants with regulatory mutations of the glutamate dehydrogenase gene.

Authors:  C A Stanley; Y K Lieu; B Y Hsu; A B Burlina; C R Greenberg; N J Hopwood; K Perlman; B H Rich; E Zammarchi; M Poncz
Journal:  N Engl J Med       Date:  1998-05-07       Impact factor: 91.245

3.  Hepatocyte nuclear factor 4α gene mutation associated with familial neonatal hyperinsulinism and maturity-onset diabetes of the young.

Authors:  Mia M Pingul; Nkecha Hughes; Anthony Wu; Charles A Stanley; Philip A Gruppuso
Journal:  J Pediatr       Date:  2011-02-24       Impact factor: 4.406

4.  Insights into the biochemical and genetic basis of glucokinase activation from naturally occurring hypoglycemia mutations.

Authors:  Anna L Gloyn; Kees Noordam; Michèl A A P Willemsen; Sian Ellard; Wayne W K Lam; Ian W Campbell; Paula Midgley; Chyio Shiota; Carol Buettger; Mark A Magnuson; Franz M Matschinsky; Andrew T Hattersley
Journal:  Diabetes       Date:  2003-09       Impact factor: 9.461

5.  Clinical characteristics and biochemical mechanisms of congenital hyperinsulinism associated with dominant KATP channel mutations.

Authors:  Sara E Pinney; Courtney MacMullen; Susan Becker; Yu-Wen Lin; Cheryl Hanna; Paul Thornton; Arupa Ganguly; Show-Ling Shyng; Charles A Stanley
Journal:  J Clin Invest       Date:  2008-08       Impact factor: 14.808

6.  Variable effects of maturity-onset-diabetes-of-youth (MODY)-associated glucokinase mutations on substrate interactions and stability of the enzyme.

Authors:  Y Liang; P Kesavan; L Q Wang; K Niswender; Y Tanizawa; M A Permutt; M A Magnuson; F M Matschinsky
Journal:  Biochem J       Date:  1995-07-01       Impact factor: 3.857

7.  The second activating glucokinase mutation (A456V): implications for glucose homeostasis and diabetes therapy.

Authors:  Henrik B T Christesen; Bendt B Jacobsen; Stella Odili; Carol Buettger; Antonio Cuesta-Munoz; Torben Hansen; Klaus Brusgaard; Ornella Massa; Mark A Magnuson; Chiyo Shiota; Franz M Matschinsky; Fabrizio Barbetti
Journal:  Diabetes       Date:  2002-04       Impact factor: 9.461

8.  SIFT web server: predicting effects of amino acid substitutions on proteins.

Authors:  Ngak-Leng Sim; Prateek Kumar; Jing Hu; Steven Henikoff; Georg Schneider; Pauline C Ng
Journal:  Nucleic Acids Res       Date:  2012-06-11       Impact factor: 16.971

Review 9.  The Genetic and Molecular Mechanisms of Congenital Hyperinsulinism.

Authors:  Sonya Galcheva; Hüseyin Demirbilek; Sara Al-Khawaga; Khalid Hussain
Journal:  Front Endocrinol (Lausanne)       Date:  2019-02-26       Impact factor: 5.555

10.  Unravelling the genetic causes of mosaic islet morphology in congenital hyperinsulinism.

Authors:  Jayne Al Houghton; Indraneel Banerjee; Guftar Shaikh; Shamila Jabbar; Thomas W Laver; Edmund Cheesman; Amish Chinnoy; Daphne Yau; Maria Salomon-Estebanez; Mark J Dunne; Sarah E Flanagan
Journal:  J Pathol Clin Res       Date:  2019-10-29
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